1) Erythrocytes-cholinesterase
红细胞胆碱酯酶
2) erythrocyte acetylcholinesterase
红细胞乙酰胆碱酯酶
1.
Objective A simple,kinetic method was developed for determination of erythrocyte acetylcholinesterase(AChE),and it is suitable for automation in the routine clinical laboratory.
目的 建立一种新的连续监测红细胞乙酰胆碱酯酶 ( ACh E)自动化方法。
3) Erythrocyte-acetylcholinesterase activity
红细胞乙酰胆碱酯酶活性
4) erythrocyte esterase
红细胞酯酶
1.
The erythrocyte esterase(Es-Ⅰ and Es-Ⅲ)and plasma esterase(Es-3 and Es-4)were polymorphic.
结果表明:红细胞酯酶Es-Ⅰ、Es-Ⅲ和血浆酯酶Es-3、Es-4均存在遗传多态性,其中Es-Ⅰ和Es-Ⅲ、Es-3分别受常染色体上的一对等位基因A和B控制,各出现3种表型A、AB和B。
2.
The erythrocyte esterase Es- Ⅰ、Es-Ⅲ and plasma esterase Es-3 、 Es-4 were identified to be polymorphic,there were three different phenotypes A,AB and B controlled by 2 autosomal codominant alleles A and B at each locus in erythrocyte esterase Es- Ⅰ、 Es-Ⅲ and plasma esterase Es-3,there were two phenotypes, dominant and recessive band ,w.
结果表明红细胞酯酶Es-Ⅰ、 Es-Ⅲ和血浆酯酶Es-3、Es-4均存在遗传多态性,其中红细胞酯酶Es-Ⅰ和Es-Ⅲ以及血浆酯酶Es-3各出现3种表型A、AB和B,分别受常染色体上的一对等位基因A和B控制。
5) cholinesterase
[英][,kɔli'nestəreis] [美][,kolə'nɛstə,res, -,rez]
胆碱酯酶
1.
Study on the detection of pesticide residues in vegetable with cholinesterase extracted from wheat;
小麦中胆碱酯酶的提取及其在蔬菜农药残留检测中的应用研究
2.
Study on screening of cholinesterase for the rapid detection of pesticide residues;
农药残留快速检测用酶——胆碱酯酶的筛选
3.
Neuroprotective effects of liquiritin and its inhibitory actions on cholinesterase activity;
甘草苷的神经保护及对胆碱酯酶的抑制作用
6) ChE
胆碱酯酶
1.
The evaluation of detecting ADA and CHE for acute hepatitis;
腺苷脱氨酶、胆碱酯酶检测在急性肝炎中的应用评价
2.
Objective To explore the relationship of serum Choline Esterase(ChE),Triglycerides (TG), Cholesterol(Ch),Alanine Aminotrasferase(ALT) ,γ-Glutamyl Transpeptidase (γ-GT) etc.
目的 :探讨血清甘油三酯 (TG)、总胆固醇 (CHOL )的浓度和丙氨酸氨基转氨酶 (AL T)、γ-谷氨酰转移酶(GGT)、胆碱酯酶 (Ch E)的活性与脂肪肝的关系。
3.
The models of the human brain AChE and human serum BuChE have been constructed on the basis of the putative three-dimensional structural similarity with Torpedo california AChE.
本文以同源的电鳐胆碱酯酶（Ｔ。
补充资料：红细胞葡萄糖-6-磷酸脱氢酶缺陷
红细胞葡萄糖-6-磷酸脱氢酶缺陷
G-6-PD缺陷为X连锁不完全显性遗传，故男性发病率显著高于女性。临床上主要引起溶血性贫血。其机制是G-6-PD系磷酸戊糖途径所必需的脱氢酶，当其缺陷时，NADpH的生成不足，红细胞GSH含量减少，使红细胞膜发生改变及氧化产物(GSS-Hb、MbH）在红细胞内推积形成不可溶性变性珠蛋白小体，使红细胞变硬，可塑性减少而致溶血。G-6-PD缺陷者由于变异类型和(或）酶缺陷程度的不同可分为四种临床类型：①蚕豆病；②药物、感染性溶血性贫血；③新生儿G-6-PD缺陷溶血症；④先天性非球形细胞溶血性贫血(Ⅰ型）。
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
参考词条