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1)  DNA repair gene
DNA修复基因
1.
Study on polymorphisms in metabolic enzyme genes,DNA repair genes and individual susceptibility to hepatocellular carcinoma;
毒物代谢酶、DNA修复基因多态与肝癌遗传易感性
2.
Damage of ionizing radiation and DNA repair gene;
电离辐射损伤与DNA修复基因
3.
Meta analysis on DNA repair gene XPC single neucleotide polymorphisms and bladder cancer
DNA修复基因XPC单核苷酸多态性与膀胱癌易感性的meta分析
2)  DNA repair genes
DNA修复基因
1.
The difference in DNA repair capacity among persons depends on the polymorphisms of DNA repair genes.
机体间存在DNA修复能力(DNA repair capacity,DRC)的差异主要是取决于DNA修复基因的多态性。
2.
BACKGROUND AND AIM:To examine the mutation of DNA repair genes of human bronchial epithelial cells malignant transformation induced by glycidyl methacrylate.
背景与目的:研究甲基丙烯酸环氧丙酯(glycidyl methacrylate,GMA)致人支气管上皮(16HBE)恶性转化细胞DNA修复基因点突变情况。
3.
Relationship between polymorphisms of DNA repair genes and esophageal cancer susceptibility;
目的研究DNA修复基因-着色性干皮病基因A(XPA)、着色性干皮病基因D(XPD)、X线修复交叉互补基因1(XRCC1)和X线修复交叉互补基因3(XRCC3)多态性与食管癌易感性的关系。
3)  DNA mismatch repair gene
DNA错配修复基因
1.
Objective To observe the expression of DNA mismatch repair gene hMSH2 mRNA in esophageal cancer tissues.
目的观察食管癌组织中DNA错配修复基因hMSH2的表达。
2.
As the development of immunology and molecular biology,gene has become the focus of attention in the incidence,development and prognosis of endometrial carcinoma,such as oncogenes(ras,bcl-2,cerbB-2,β-catenin,and C-met),cancer suppressor genes(PTEN,P53,P16,and RB) and DNA mismatch repair gene.
如癌基因(ras、bcl-2、cerbB-2、β-catenin、C-met)、抑癌基因(PTEN、P53、P16、RB)及DNA错配修复基因。
4)  DNA repair gene XPG
DNA修复基因XPG
5)  DNA repair related genes
DNA修复相关基因
1.
Objective To compare gene expression gene profilie of nasopharyngeal c arcinoma(NPC) tissue with that of normal nasopharyngeal tissues by cDNA array a nd to discuss possible functions of DNA repair related genes in NPC tissue.
结论DNA修复相关基因的改变可能在鼻咽癌病理生理过程中起一定作用。
6)  DNA damage repair gene
DNA损伤修复基因
1.
Multiple myeloma (MM) is an incurable disease now, to which increasing DNA damage repair gene expression, increasing tumor cell drug resistance, and decreasing tumor cell apoptosis may contribute much.
目前,多发性骨髓瘤(MM)仍是一种不可治愈的疾病,其中重要的原因是MM细胞DNA损伤修复基因表达上调,导致其耐药增加,细胞凋亡减少。
补充资料:DNA修复
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性质:可以降低DNA碱基序列因遭受辐射和其他诱变因子所致损伤的一种机制。此过程是一种特异酶[包括DNA聚合酶(DNA连接酶)]催化反应。DNA修复系统保证了遗传有效突变率大大低于始发突变率。DNA修复是一由多种不同机制参与的复杂过程。有些在光照时增强,有些则需在无光条件下进行。

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