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1)  hypoxic pulmonary vascular remolding
缺氧肺血管重构
2)  pulmonary vascular remodeling
肺血管重构
1.
Role of bcl-2 and bax expressions in pulmonary vascular remodeling of PAH rats secondary to left-right shunt CHD
bcl-2/bax表达在左向右分流型肺动脉高压肺血管重构中的作用
2.
Objective To explore the relation between the expression of AT1 or AT2 receptors and pulmonary vascular remodeling.
目的探讨左向右分流先心病患儿肺血管AT1、AT2受体表达与肺血管重构的关系。
3.
Objective: Based on the observation on effect of ACEI and AT_1 receptor antagonist on pulmonary vascular remodeling in rats with pulmonary hypertension, the mechanism of AngⅡrole in the development of pulmonary vascular remodeling were explored.
目的:观察ACEI和AT_1受体拮抗剂对大鼠肺切除加野百合碱诱导肺动脉高压肺血管重构的影响,进而探讨AngⅡ在肺动脉高压肺血管重构的机制。
3)  pulmonary vascular remodeling
肺血管结构重建
1.
Effects of Histamine on Pulmonary Vascular Remodeling in Broiler Chickens Induced by Low Ambient Temperature;
组胺在低温诱发的肉鸡肺血管结构重建中的作用
2.
Objectives To investigate whether pulmonary vascular remodeling in rats with hypoxic pulmonary hypertension(HPH)could be prevented by treatment with Rho kinase inhibitor fasudil.
目的探讨Rho激酶抑制剂法舒地尔对大鼠低氧性肺动脉高压(HPH)及其肺血管结构重建的影响。
4)  pulmonary vascular remodeling(PVR)
肺血管构型重建
1.
The pulmonary vascular remodeling(PVR)in hypoxia was based on the pathological development of the hypoxic pulmonary hypertension (HPH).
本文认为,缺氧性肺血管构型重建是低氧性肺动脉高压持续发展的主要病理基础,通过对临床和病理改变的分析,提出形成肺血管构型重建的中医病因病机为:气虚是始动因素,又是基本病机,是肺气虚向肺肾气虚的演变,血瘀、痰阻是继发的病理改变,肺络不通是局部的病理结果;治疗应以补益肺肾、活血消痰、疏通肺络为基本原则。
5)  Hypoxic Pulmonary Vasoconstriction
缺氧性肺血管收缩
1.
Role of the Rho/Rho-kinase pathway in the mechanism of hypoxic pulmonary vasoconstriction
Rho/Rho激酶信号通路在缺氧性肺血管收缩中的机制
6)  acute hypoxic pulmonary vasoconstriction
急性缺氧性肺血管收缩
1.
Study on the roles of voltaged potassium channels in acute hypoxic pulmonary vasoconstriction;
K~+v通道亚型在急性缺氧性肺血管收缩中的作用研究
补充资料:肺错构瘤


肺错构瘤
pulmonary hemartoma

肺正常组织在胚胎发育过程中,过度生长形成的瘤样畸形。为良性肿瘤,极少恶变。主要由软骨形成,内含有上皮、平滑肌组织、纤维组织和脂肪等。多位于肺边缘,呈孤立性结节,少数发生于气管内。多数无症状,仅在X线检查时被发现。肿瘤较大且靠近肺门,可有咳嗽、气急和胸痛等症状。治疗主要是肺叶切除。
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