1) Pulmonary vascular collagen remodeling
肺血管胶原重塑
2) Pulmonary vascular remodeling
肺血管重塑
1.
Pulmonary vascular remodeling was observed with optical microscope, and HIF-α mRNA and protein were detected by in situ hybridization and immunohistochemistry .
目的通过观察慢性阻塞性肺疾病(COPD)患者肺小动脉内低氧诱导因子(HIF)-α的表达,探讨其在肺血管重塑中的可能作用。
2.
Pulmonary vascular remodeling and proliferation of vascular smooth muscle cells (VSMCs) of pulmonary arteries of broilers in the development of ascites syndrome(AS) induced by low ambient temperature were dynamically analyzed to study the mechanism of pulmonary vascular remodeling.
本试验旨在研究低温诱发肉鸡腹水综合征(AS)过程中平滑肌细胞增殖在肺血管重塑过程中的动态变化,初步探讨肺血管重塑的机制。
3) pulmonary vascular structural remodeling
肺血管重塑
1.
The effect of BQ123,a selective ETA-receptor antagonist, on pulmonary vascular structural remodeling induced by low ambient temperature was investigated through observing the dynamic change of pulmonary arterioles medial thickness and pulmonary blood vessel endometry area caused by proliferous smooth muscle cells in broiler chickens.
动态观察了肺小动脉中膜平滑肌的增殖及管腔面积的改变,探讨内皮素受体拮抗剂BQ123 对低温诱发的肉鸡肺血管重塑的影响。
4) collagen remodeling
胶原重塑
1.
Allopurinol attenuates collagen remodeling of non-infarcted myocardium after myocardial infarction in rats;
别嘌呤醇降低大鼠心肌非梗死区胶原重塑
5) Vascular remodeling
血管重塑
1.
Study on correlation between vascular remodeling in essential hypertension and collateral disease;
高血压血管重塑与络病的相关性
2.
Effect of simvastatin on vascular remodeling and TGF-β_1 expression in vascular adventitia in carotids of rats with ballon injury;
辛伐他汀对球囊损伤颈动脉后大鼠血管重塑及血管外膜转化生长因子-β_1表达的影响
3.
Effects of intimal hyperplasia and vascular remodeling on vascular restenosis after balloon injury;
血管球囊损伤后内膜增生及血管重塑对再狭窄的影响
6) pulmonary vascular remodeling
肺血管重构
1.
Role of bcl-2 and bax expressions in pulmonary vascular remodeling of PAH rats secondary to left-right shunt CHD
bcl-2/bax表达在左向右分流型肺动脉高压肺血管重构中的作用
2.
Objective To explore the relation between the expression of AT1 or AT2 receptors and pulmonary vascular remodeling.
目的探讨左向右分流先心病患儿肺血管AT1、AT2受体表达与肺血管重构的关系。
3.
Objective: Based on the observation on effect of ACEI and AT_1 receptor antagonist on pulmonary vascular remodeling in rats with pulmonary hypertension, the mechanism of AngⅡrole in the development of pulmonary vascular remodeling were explored.
目的:观察ACEI和AT_1受体拮抗剂对大鼠肺切除加野百合碱诱导肺动脉高压肺血管重构的影响,进而探讨AngⅡ在肺动脉高压肺血管重构的机制。
补充资料:胶原三重螺旋体
分子式:
CAS号:
性质: 又称原胶原(tropocollagen)。是胶原分子的组成单元。原胶原呈细长的棒状结构,长约3000nm,直径约1.5nm,分子量为30万左右,由三条等长的α-多肽链(α链)组成。每条α链先自身左旋(螺距约0.95nm),然后相互缠绕(似电缆)形成右旋超螺旋体。螺旋体的主体部分为规整结构,其两端为伸展部分,即氨基端(5.0hm)和羧基端(7.5nm),它们又被称为肽尾(telopeptides),(图暂缺)。胶原三重螺旋体应是确指原胶原规整结构的主体部分,它们对化学试剂、酶等生物制品有很高的稳定性。
CAS号:
性质: 又称原胶原(tropocollagen)。是胶原分子的组成单元。原胶原呈细长的棒状结构,长约3000nm,直径约1.5nm,分子量为30万左右,由三条等长的α-多肽链(α链)组成。每条α链先自身左旋(螺距约0.95nm),然后相互缠绕(似电缆)形成右旋超螺旋体。螺旋体的主体部分为规整结构,其两端为伸展部分,即氨基端(5.0hm)和羧基端(7.5nm),它们又被称为肽尾(telopeptides),(图暂缺)。胶原三重螺旋体应是确指原胶原规整结构的主体部分,它们对化学试剂、酶等生物制品有很高的稳定性。
说明:补充资料仅用于学习参考,请勿用于其它任何用途。
参考词条