1) pulmonary intravascular macrophages
肺血管内巨噬细胞
1.
Objective To explore the role of pulmonary intravascular macrophages(PIM)in the pathogenesis of hepatopulmonary syndrome(HPS).
目的探讨肺血管内巨噬细胞(pulmonary intravascular macrophages,PIM)在肝肺综合征(hepatopulmonary syndrome,HPS)发病机制中的作用。
2) Pulmonary intravascular macrophage accumulation
肺血管内巨噬细胞聚积
3) alveolar macrophages
肺巨噬细胞
1.
Alveolar macrophages of Sprague_Dawley rat were served in vitro as target cells to observe the effects on the phagocytosis of alveolar macrophages and the levels of TNF_α after exposure to PM10 and airborne fungi either alone or both.
用不同浓度的PM10、气传真菌 ,以及PM10 和气传真菌联合染毒液染毒大鼠肺泡巨噬细胞 ,分别测定培养液中肺泡细胞存活率、吞噬率和吞噬指数以及细胞因子TNF_α ,观察PM10 与气传真菌单独和联合染毒对肺巨噬细胞存活率及其吞噬功能的影响。
4) alveolar macrophage
肺泡巨噬细胞
1.
Study on cytotoxicity of nanosized silica and surface modified nanosized silica in rat alveolar macrophages;
未修饰纳米SiO_2和表面修饰型纳米SiO_2对大鼠肺泡巨噬细胞的毒性研究
2.
Expression of Toll-like receptor 2/4 on alveolar macrophage in the model of total hepatic ischemia/reperfusion injury in mice;
全肝缺血再灌注小鼠肺泡巨噬细胞Toll样受体2/4的表达
3.
Effect of sodium selenite and magnesium sulfate in antagonism to the oxidative injury of silica in alveolar macrophages;
硒和镁拮抗二氧化硅致肺泡巨噬细胞氧化损伤的研究
5) alveolar macrophages
肺泡巨噬细胞
1.
Impact of dust storm PM_(2.5)and PM_(10) on the phagocytic function of alveolar macrophages of rat;
沙尘暴PM_(2.5)、PM_(10)对大鼠肺泡巨噬细胞吞噬功能的影响
2.
Damage effects of sulfur dioxide inhalation on DNA damages in alveolar macrophages from male rats;
二氧化硫对大鼠肺泡巨噬细胞DNA的损伤作用
3.
DATS inhibited LPS-induced proinflammatory cytokines expression in mouse alveolar macrophages cell line MH-S by inhibiting NF-κB activation;
DATS通过NF-κB抑制LPS诱导小鼠肺泡巨噬细胞促炎细胞因子表达
6) pulmonary alveolar macrophages
肺泡巨噬细胞
1.
Study on the mechanism of industrial mineral dusts effect on pulmonary alveolar macrophages injury;
工业矿物粉尘对肺泡巨噬细胞影响的体外研究
2.
Effects of nitric oxide on activation of nuclear factor-κB in pulmonary alveolar macrophages in lipopolysac charide-induced acute lung injury;
一氧化氮对内毒素刺激肺泡巨噬细胞核因子-κB的影响
3.
To investigate the transdifferentiation of pulmonary artery endothelium cells to smooth muscle cells under hypoxia and the role of pulmonary alveolar macrophages(PAM) in this process.
研究低氧条件下肺动脉内皮细胞(PAEC)向平滑肌样细胞的转分化及肺泡巨噬细胞(PAM)在此转分化过程中的作用。
补充资料：内噬
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