Porcine alveolar macrophage (PAM) is both an important target cell of PCV2 and an important immune cell.

Porcine pulmonary alveolar macrophages(PAMs)were infected with A/Swine/Guangdong/1/2004,a swine influenza virus(SIV)isolate.

Study on cytotoxicity of nanosized silica and surface modified nanosized silica in rat alveolar macrophages;

Expression of Toll-like receptor 2/4 on alveolar macrophage in the model of total hepatic ischemia/reperfusion injury in mice;

Effect of sodium selenite and magnesium sulfate in antagonism to the oxidative injury of silica in alveolar macrophages;

Impact of dust storm PM_(2.5)and PM_(10) on the phagocytic function of alveolar macrophages of rat;

Damage effects of sulfur dioxide inhalation on DNA damages in alveolar macrophages from male rats;

DATS inhibited LPS-induced proinflammatory cytokines expression in mouse alveolar macrophages cell line MH-S by inhibiting NF-κB activation;

Study on the mechanism of industrial mineral dusts effect on pulmonary alveolar macrophages injury;

Effects of nitric oxide on activation of nuclear factor-κB in pulmonary alveolar macrophages in lipopolysac charide-induced acute lung injury;

To investigate the transdifferentiation of pulmonary artery endothelium cells to smooth muscle cells under hypoxia and the role of pulmonary alveolar macrophages(PAM) in this process.

Extracting,purifying and activity testing of pulmonary alveolar macrophages of mice;

Effects of PRRSV and PCV2 co-infection on the transcription of CD80-CD86 mRNAs in porcine pulmonary alveolar macrophage;

In order to study the damaging mechanism of pulmonary alveolar macrophages, the changes of their death ratio,malandialdthyde (MDA) content and activities of dehydrogenate (LDH) and superoxide dismitase (SOD) were determined, and the technique of VITRO cell culture was used to investigate the cytotoxicity of dusts of six minerals (twelve cryst habits) from twelve mineral deposits.