1) c-Jun N-terminal proteinkinase (JNK)
c-Jun氨基端激酶(JNK)
2) c-Jun N-terminal kinase(JNK)
c-Jun氨基末端激酶(JNK)
3) JNK
c-Jun氨基端激酶
1.
[Methods] 32 SD rats were divided equally into four groups,sham group,ischemia reperfusion group,curcumine group and control group,The number of cells in cerebrum hippocampus CA1 were detected by cresyl staining method,the activation of c-Jun femical kinase(JNK) was analyzed by immunoblotting.
用焦油紫染色法检测其大脑海马CA 1区的细胞数量,免疫印迹法分析c-Jun氨基端激酶(JNK)的激活情况。
2.
Objective:The present study aimed at investigating etoposide-induced apoptosis and the underlying mechanisms involving activation of c-Jun NH2-terminal kinase (JNK) and caspase-3 using the human leukemia cell line HL-60 cells.
目的:以人白血病HL-60细胞为实验,研究Etoposide(依托泊苷)诱导HL-60细胞凋亡, Caspase-3(Cysteinyl aspartate-specific proteinase-3,半胱天冬酶-3)、JNK(c-Jun NH2-terminal kinase ,c-Jun氨基端激酶)活性变化及Caspase抑制剂z-VAD-FMK对酶活性的影响, 探讨其可能的作用机制。
4) C-Jun N-terminal kinase
C-Jun氨基端激酶
5) c-jun NH2-terminal kinase
c-Jun氨基端激酶
1.
Compared with the control group,the impact of AEBR on insulin sensitivity and activation of c-Jun NH2-terminal kinase(JNK) in fructose-fed rats supplemented with or without 5 g/kg of AEBR was assessed.
目的观察黑米花色苷提取物(AEBR)对果糖喂养大鼠胰岛素敏感性的影响,并从c-Jun氨基端激酶(JNK)活化程度的变化分析其作用机制。
6) JNK
c-jun氨基末端激酶
1.
Objective:To study the action mechanism of p,p'-DDE and /or β-BHC on JNK and MAPK signal transduction pathways in rat Sertoli cells in vitro.
目的:研究2,2-双-(对氯苯基)-1,1-二氯乙烯(p,p'-DDE)和β-六氯化苯(β-BHC)单独作用及联合作用对离体培养大鼠睾丸Sertoli细胞c-jun氨基末端激酶(JNK)丝裂原活化蛋白激酶(MAPK)信号转导通路的作用机制。
2.
However, it remains largely controversial whether c-Jun NH2- terminal kinase (JNK) is involved in the effects of insulin on MI/R injury.
然而c-Jun氨基末端激酶(c-Jun NH2-terminal kinase,JNK)信号通路在胰岛素保护I/R心肌中的作用尚不清楚,本研究旨在探讨JNK信号通路在胰岛素保护I/R心肌中的作用及其与PI3-K/Akt信号通路间的相互关系。
3.
Objective:To study the action mechanism of p,p -DDE and /or β-BHC on JNK and MAPK signal transduction pathways in rat Sertoli cells in vitro.
目的:研究2,2-双-(对氯苯基)-1,1-二氯乙烯(p,p'-DDE)和β-六氯化苯(β-BHC)单独作用及联合作用对离体培养大鼠睾丸Sertoli细胞c-jun氨基末端激酶(JNK)丝裂原活化蛋白激酶(MAPK)信号转导通路的作用机制。
补充资料：[3-(aminosulfonyl)-4-chloro-N-(2.3-dihydro-2-methyl-1H-indol-1-yl)benzamide]
分子式：C16H16ClN3O3S
分子量：365.5
CAS号：26807-65-8
性质：暂无
制备方法：暂无
用途：用于轻、中度原发性高血压。
分子量：365.5
CAS号：26807-65-8
性质：暂无
制备方法：暂无
用途：用于轻、中度原发性高血压。
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
参考词条