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1)  Inhibitor of nuclear factor-KBa (IkBa)
c-Jun-N末端激酶
2)  c-Jun N-terminal kinase
c-Jun N-末端激酶
1.
Synergistic effect of thermotherapy in combination with chemotherapy on lung tumor A549 cells growth through activation of c-Jun N-terminal kinase and inhibition of heat shock protein70 expression;
热化联合对肺癌A549细胞生长、c-Jun N-末端激酶磷酸化及热休克蛋白70表达的影响
3)  JNK
c-jun氨基末端激酶
1.
Objective:To study the action mechanism of p,p'-DDE and /or β-BHC on JNK and MAPK signal transduction pathways in rat Sertoli cells in vitro.
目的:研究2,2-双-(对氯苯基)-1,1-二氯乙烯(p,p'-DDE)和β-六氯化苯(β-BHC)单独作用及联合作用对离体培养大鼠睾丸Sertoli细胞c-jun氨基末端激酶(JNK)丝裂原活化蛋白激酶(MAPK)信号转导通路的作用机制。
2.
However, it remains largely controversial whether c-Jun NH2- terminal kinase (JNK) is involved in the effects of insulin on MI/R injury.
然而c-Jun氨基末端激酶(c-Jun NH2-terminal kinase,JNK)信号通路在胰岛素保护I/R心肌中的作用尚不清楚,本研究旨在探讨JNK信号通路在胰岛素保护I/R心肌中的作用及其与PI3-K/Akt信号通路间的相互关系。
3.
Objective:To study the action mechanism of p,p -DDE and /or β-BHC on JNK and MAPK signal transduction pathways in rat Sertoli cells in vitro.
目的:研究2,2-双-(对氯苯基)-1,1-二氯乙烯(p,p'-DDE)和β-六氯化苯(β-BHC)单独作用及联合作用对离体培养大鼠睾丸Sertoli细胞c-jun氨基末端激酶(JNK)丝裂原活化蛋白激酶(MAPK)信号转导通路的作用机制。
4)  c-Jun N-terminal kinase
c-Jun氨基末端激酶
1.
Role of c-Jun N-terminal kinase in the regulation of TGF-β_1-induced fibronectin production in rat mesangial cells;
c-Jun氨基末端激酶在转化生长因子-β_1诱导肾系膜细胞合成纤维连接蛋白中的作用
2.
Expression and significance of c-Jun N-terminal kinase (JNK) in the rat of nonalcoholic fatty liver disease with insulin resistance
c-jun氨基末端激酶在非酒精性脂肪肝病胰岛素抵抗大鼠中的表达及意义
3.
Objective:To study the expression of phosphorylated c-Jun N-terminal kinase (P-JNK) in lung tissues,the amounts of IL-1β in serum and BALF of asthmatic rats of airway remodeling,whereby to explore the role of P-JNK in asthma airway remodeling and the possible effect of IL-1β on JNK activation.
目的:研究哮喘气道重塑大鼠肺组织磷酸化c-Jun氨基末端激酶(P-JNK)的表达,血清/支气管肺泡灌洗液(BALF)白介素1β(IL-1β)的含量,探讨P-JNK在哮喘气道重塑中的作用及IL-1β对JNK活化的可能影响。
5)  C-jun N-terminal kinase(JNK)
C-jun氨基末端激酶
1.
Objective To explore the role of c-Jun N-terminal kinase(JNK)in the apoptosis induced by apoptin gene in human melanoma cell A375.
目的研究肿瘤特异性凋亡基因(Apoptin gene)在诱导人黑色素瘤细胞A375发生凋亡时,c-Jun氨基末端激酶(JNK)信号通路在细胞发生凋亡中的作用。
6)  c-Jun amino-terminal kinase
c-Jun 氨基末端激酶
1.
Expression of α-smooth muscle actin(α-SMA),secretion of fibronectin (FN)and c-Jun amino-terminal kinase(JNK)activity were assayed by Western blot and ELISA,the change of distribu- tion of α-SMA in mesangial cells before and after IL-1β was ob.
方法在体外培养的大鼠肾小球系膜细胞中,以IL-lβ和 AⅡ为刺激物,采用蛋白印记和酶联免疫吸附法测定α-平滑肌动蛋白(a-SMA)表达、纤维粘连蛋白(FN)分泌量和 c-Jun 氨基末端激酶(JNK)活性,用共聚焦显微镜观察 IL-lβ刺激前后系膜细胞内α-SMA 的分布变化,并观察 A&A 血清滤液对上述指标的影响。
补充资料:长末端重复
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性质:复合转座子含有一个中心序列和位于其两侧的臂(arm)。中心序列含有抗药性基因、转座酶基因等遗传信息,左、右两臂在序列上相似,有的就是相同的插入序列,例如Tn9的两臂就是ISI。大多数情况下两臂的方向是相反的。

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