1) pain and hyperalgesia
痛及痛过敏
1.
The release of excitatory amino acids(EAAs) and up-regulation or increase in activation of EAAs receptor in pathway of nociceptive information can contribute to pain and hyperalgesia.
痛及痛过敏的形成与脊髓水平伤害信息传递通路中初级传入纤维(A_δ、C纤维)末梢释放的兴奋性氨基酸(excitator amino acids，EAAs)及其受体的活动增强及表达上调有关。
2) inflammatory pain and hyperalgesia
炎性痛及痛过敏
1.
Aim: To observe the changes of cyclooxygenase-2 (COX-2) expression and especially the time course of the changes in dorsal horn of the spinal cord during formalin-induced inflammatory pain and hyperalgesia in rats.
目的 :观察甲醛炎性痛及痛过敏过程中脊髓后角环氧化酶 2 (COX 2 )表达的变化及其时间特征。
4) hyperalgesia
[英][,haipəræl'dʒi:ziə] [美][,haɪpəræl'dʒizɪə]
痛觉过敏
1.
Relationship between Abnormal spinal complement activity and hyperalgesia in rats following sciatic nerve ligation;
坐骨神经结扎大鼠脑脊液补体异常活化与痛觉过敏的关系
2.
Intrathecal minocycline attenuates thermal hyperalgesia in a rat model of CFA-induced arthritis;
鞘内注射米诺环素减轻大鼠佐剂性关节炎性痛觉过敏
3.
Effects of NMDA receptors on hyperalgesia;
NMDA受体在痛觉过敏中的作用
5) hyperalgesia
[英][,haipəræl'dʒi:ziə] [美][,haɪpəræl'dʒizɪə]
痛过敏
1.
The relieving effect of cyclooxygenase inhibitors and prostaglandin receptor blocker on hyperalgesia of diabetic rats;
环加氧酶抑制剂和前列腺素受体阻断剂缓解糖尿病大鼠的痛过敏
2.
Using NADPH d histochemistory, we observed the changes of nitric oxide synthase (NOS), especially in its time course, in the dorsal horn of the spinal cord during formalin induced inflammatory pain and hyperalgesia.
用NADPH 黄递酶 (NADPH d)组化法观察福尔马林炎症性痛及痛过敏过程中脊髓后角一氧化氮合酶 (NOS)的变化 ,特别是其时间特征。
3.
Intradental hyperalgesia is of complicated mechanism, which is not clearly understood.
本研究采用细胞内Ca~(2+)浓度测定技术、免疫组织化学技术等，系统观察了牙髓炎症性痛过敏状态下神经纤维内Ca~(2+)浓度及神经肽的变化；三叉神经节中神经肽及一氧化氮合酶的变化；三叉神经脊柬核尾侧亚核c—fos蛋白的表达及一氧化氮合酶的变化。
6) heat-hyperalgesia
热痛过敏
1.
Results:Bupivacaine could completely inhibit heat-hyperalgesia, inhi.
结果 :①行为学测定 :实验组大鼠始终未出现热痛过敏症状 ,而对照组大鼠出现了持续的热痛过敏症状。
补充资料：反射痛
反射痛
reflex pain
牵涉痛的一种形式，某些脏器病变发生的疼痛放射到某处所引起的疼痛。如胆绞痛可反射性引起右肩背部痛、肾绞痛沿输尿管放射到会阴部等等。
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
参考词条