1)  glucose deprivation
缺糖
1.
Objective:To probe into the pathological mechanism of the injury induced by glucose deprivation(an insult relevant to the brain ischemia) in PC12 cell line,providing a basis for studying the mechaism for post-eschemia neuron damage.
目的:研究PC12细胞缺糖损伤的分子生物学特征。
2)  hypoxia/hypoglycemia
缺氧/缺糖
1.
Protective effects of muscone(麝香酮)on the hypoxia/hypoglycemia and reoxygenation induced damage of SH-SY5Y nerve cell cultured in vitro;
麝香酮对SH-SY5Y神经细胞缺氧/缺糖和再给氧损伤的保护作用
2.
Protective effects of Shenmai Injection and ginsenoside Rb_1 and Rg_1 on damage of neurons, VEC and astrocytes induced by hypoxia/hypoglycemia and reoxygenation;
参麦注射液及人参皂苷Rb_1、Rg_1抗神经、血管内皮、星形胶质细胞缺氧/缺糖损伤的研究
3.
Protective effects of salidroside on injury induced by hypoxia/hypoglycemia in cultured neurons;
红景天苷对缺氧/缺糖损伤神经细胞的保护作用
3)  Oxygen-glucose deprivation
缺氧缺糖
1.
Overexpression of PDZ1 domain of PSD-95 protein rescues hippocampl neurons from apoptosis induced by oxygen-glucose deprivation;
PSD-95结构域PDZ1过表达拮抗缺氧缺糖诱导的海马神经元凋亡
2.
Protection of rhodioside derivative S01 against damage of SH-SY5Y cells induced by glutamate and oxygen-glucose deprivation;
红景天苷衍生物S01对谷氨酸及缺氧缺糖所致SH-SY5Y细胞损伤的保护作用
3.
In this study, the effect of oxygen-glucose deprivation (OGD) on the cellular production of NO was investigated in cultured hippocampal neurons.
研究了原代培养的海马神经细胞中 ,缺氧缺糖对NO合成的影响。
4)  oxygen-glucose deprivation
缺糖、缺氧
1.
Aim:To observe the protective effects of protein transduction domain-kallikrein(PTD-kallikrein)on neurons incubated in condition of oxygen-glucose deprivation(OGD)/recovery,and to explore the protective mechanism.
目的:观察蛋白质转导域-激肽释放酶(PTD-kallikrein)对缺糖、缺氧(OGD)再复糖、复氧培养的神经元保护作用,探讨其可能的作用机制。
5)  Oxygen-glucose deprivation
缺糖缺氧
1.
The protective effect of LY367385 on impairment of cultured mouse cerebral cortical neurons induced by sodium glutamate or oxygen-glucose deprivation;
LY367385对谷氨酸钠及缺糖缺氧引起的培养小鼠大脑皮层神经元损伤的保护作用
6)  glucose deprivation
缺糖损伤
1.
Glucose regulates protein 75 expression in PC12 cell line under glucose deprivation;
葡萄糖调节蛋白75在缺糖损伤PC12细胞中的表达
2.
Protective effect of glutamine on PC12 cells under glucose deprivation;
谷氨酰胺对细胞缺糖损伤的保护作用
3.
Objective To evaluate the protective effect of Salvianoic acid B(SAB) to cells under glucose deprivation and to explore the mechanism of the protective effect of SAB.
目的观察丹酚酸B(SAB)对PC12细胞缺糖损伤的作用并探讨其作用的机制。
参考词条
补充资料：阿卡波糖 ,拜糖平,拜糖苹
药物名称：拜糖平

英文名：Acarbose

别名： 阿卡波糖 ,拜糖平,拜糖苹
适应症： 本品是一种安全平稳的新型糖尿病治疗药。配合饮食治疗糖尿病。可单用或与其他降血糖药合用。
用量用法：
口服：开始每日１５０ｍｇ（指含阿卡波糖）、随后增至每日３００～６００ｍｇ。剂量可在１～２周内增加（平均剂量每日３００ｍｇ）、饭前服或与头几口食物嚼服才有效。服用疗程没有限制。
注意事项：
无全身性副反应、无低血糖反应、不影响体重。常有胀气、肠鸣；偶有腹泻、腹痛等。１８岁以下的患者、乳妇不宜用。
规格： ５０ｍｇ／片，１００ｍｇ／片（含阿卡波糖）。



类别：胰岛激素及影响血糖药
说明：补充资料仅用于学习参考，请勿用于其它任何用途。