1)  CDT
缺糖转铁蛋白
2)  glucose deprivation
缺糖
1.
Objective:To probe into the pathological mechanism of the injury induced by glucose deprivation(an insult relevant to the brain ischemia) in PC12 cell line,providing a basis for studying the mechaism for post-eschemia neuron damage.
目的:研究PC12细胞缺糖损伤的分子生物学特征。
3)  hypoxia/hypoglycemia
缺氧/缺糖
1.
Protective effects of muscone(麝香酮)on the hypoxia/hypoglycemia and reoxygenation induced damage of SH-SY5Y nerve cell cultured in vitro;
麝香酮对SH-SY5Y神经细胞缺氧/缺糖和再给氧损伤的保护作用
2.
Protective effects of Shenmai Injection and ginsenoside Rb_1 and Rg_1 on damage of neurons, VEC and astrocytes induced by hypoxia/hypoglycemia and reoxygenation;
参麦注射液及人参皂苷Rb_1、Rg_1抗神经、血管内皮、星形胶质细胞缺氧/缺糖损伤的研究
3.
Protective effects of salidroside on injury induced by hypoxia/hypoglycemia in cultured neurons;
红景天苷对缺氧/缺糖损伤神经细胞的保护作用
4)  Oxygen-glucose deprivation
缺氧缺糖
1.
Overexpression of PDZ1 domain of PSD-95 protein rescues hippocampl neurons from apoptosis induced by oxygen-glucose deprivation;
PSD-95结构域PDZ1过表达拮抗缺氧缺糖诱导的海马神经元凋亡
2.
Protection of rhodioside derivative S01 against damage of SH-SY5Y cells induced by glutamate and oxygen-glucose deprivation;
红景天苷衍生物S01对谷氨酸及缺氧缺糖所致SH-SY5Y细胞损伤的保护作用
3.
In this study, the effect of oxygen-glucose deprivation (OGD) on the cellular production of NO was investigated in cultured hippocampal neurons.
研究了原代培养的海马神经细胞中 ,缺氧缺糖对NO合成的影响。
5)  oxygen-glucose deprivation
缺糖、缺氧
1.
Aim:To observe the protective effects of protein transduction domain-kallikrein(PTD-kallikrein)on neurons incubated in condition of oxygen-glucose deprivation(OGD)/recovery,and to explore the protective mechanism.
目的:观察蛋白质转导域-激肽释放酶(PTD-kallikrein)对缺糖、缺氧(OGD)再复糖、复氧培养的神经元保护作用,探讨其可能的作用机制。
6)  Oxygen-glucose deprivation
缺糖缺氧
1.
The protective effect of LY367385 on impairment of cultured mouse cerebral cortical neurons induced by sodium glutamate or oxygen-glucose deprivation;
LY367385对谷氨酸钠及缺糖缺氧引起的培养小鼠大脑皮层神经元损伤的保护作用
参考词条
补充资料:转铁蛋白
分子式:
CAS号:

性质:又称转铁蛋白。存在于脊椎动物血液中,是铁的运载者。由β1球蛋白和Fe3+构成的一类糖蛋白,含680~700个氨基酸残基,有两条肽链,每条可结合1个Fe3+。主要功能是将铁转运到肝和脾脏以及网织红细胞。失去铁的铁传递蛋白即去辅基铁传递蛋白,是指铁蛋白的外壳。

说明:补充资料仅用于学习参考,请勿用于其它任何用途。