1) hypoxia-hypoglycemia and reoxygenation
缺氧-缺糖再给氧
1.
Objective: To investigate the protect effects of Qingkailing injection on mitochondrion membrane potential(MMP) during injury induced by hypoxia-hypoglycemia and reoxygenation in cultured rat hippocampal neurons.
结果:缺氧-缺糖5 h再给氧3 h时,细胞内[Ca2+]i和细胞凋亡率明显增加,并随再给氧时间的延长而明显增高,线粒体膜电位和活性明显降低,并随再给氧时间的延长而进一步下降,清开灵注射液能显著降低细胞内[Ca2+]i浓度,抑制细胞凋亡的发生,提高线粒体膜电位和活性,与缺氧-缺糖再给氧组相比均有显著性差异(P<0。
2) Hypoxia and hypoxia-reoxygenation
缺氧缺糖后再给氧给糖
3) hypoxia-reoxynation
缺氧再给氧
1.
Protection and its mechanism of catechin morphon on hypoxia-reoxynation induced injury in myocardial cells;
儿茶素单体对心肌细胞缺氧再给氧损伤的保护作用及机制研究
2.
Methods After establishing hypoxia-reoxynation model on myocardial cells,the content of LDH,MDA and apoptosis rate were examined.
方法在建立心肌细胞缺氧再给氧损伤模型基础上,研究川芎嗪对细胞培养液中乳酸脱氢酶及心肌细胞中丙二醛含量的影响;同时,探讨其对心肌细胞凋亡率的影响。
4) hypoxia-reoxygenation
缺氧-再给氧
1.
Effects of hyperin on myocardial cell injured by hypoxia-reoxygenation in primary cultured rat;
金丝桃苷对原代培养的大鼠心肌细胞缺氧-再给氧损伤的影响
5) hypoxia/reoxygenation
缺氧/再给氧
1.
Although we have known that the expression of death receptors for TNF-related apoptosis-inducing ligand(TRAIL)could be induced on hepatocytes,the effects of hypoxia/reoxygenation(H/R)on TRAIL-mediated apoptosis is unclear.
目的:研究肿瘤坏死因子相关凋亡诱导配体(TNF-related apoptosis-inducing ligand,TRAIL)/死亡受体5(Death receptor 5,DR5)途径在人肝细胞缺氧/再给氧(Hypoxia/reoxygenation,H/R)损伤中的作用,并探讨其作用机制。
6) oxygen-glucose deprivation/reperfusion(OGD/RP)
缺氧缺糖/再灌注
1.
OBJECTIVE To investigate the protective effect of baicalin on oxygen-glucose deprivation/reperfusion(OGD/RP)-induced injury and possible mechanism in mouse brain slices and rat cortical neurons.
目的在离体水平,探讨黄芩苷对缺氧缺糖/再灌注(OGD/RP)诱导的脑片及神经元损伤的保护作用及机制。
补充资料：缺氧缺血性脑病
缺氧缺血性脑病
hypoxic ischemic encephalopathy,HIE
发生在围产儿，尤易发生在窒息足月儿的疾病。脑组织以水肿、软化、坏死和出血为主要病变。重者有后遗症。
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
参考词条