1) MDA-MB-453
MDA-MB-453细胞
1.
Methods The inhibitory effects of Herceptin and 9-cis RA on the proliferation of MDA-MB-453 cells were detected by the MTT assay.
结果一定剂量的Herceptin和9-顺式视黄酸能够有效协同抑制MDA-MB-453细胞的增殖活性。
2.
Methods MDA-MB-453 breast cancer cells were treated with 5 μg/ml Herceptin or 1 μmol/L 9-cis-RA or both for 24, 48 or 72 h.
方法用Herceptin、9-顺式视黄酸单独和联合处理MDA-MB-453细胞24~72h,用MTT法观察其对乳腺癌细胞的增殖抑制效应,通过流式细胞仪法检测乳腺癌细胞周期相的分布,用TUNEL法检测细胞凋亡的情况。
2) human breast cancer cell line MDA-MB-453
人乳腺癌细胞MDA-MB-453
1.
Effects of genistein and chemotherapeutic agents on proliferation of human breast cancer cell line MDA-MB-453;
方法GEN和化疗药物PTX、DOX单独或联合处理体外培养的人乳腺癌细胞MDA-MB-453,MTT法测定细胞增殖抑制率并用联合用药公式分析合并效应,流式细胞仪分析细胞周期,形态学观察和Annexin-V-FITC/PI双标记法检测细胞凋亡。
3) MDA-MB-453 breast cancer cell
MDA-MB-453乳腺癌细胞
1.
Objective:To study the effects of genistein on the expression of vascular endothelial growth factor (VEGF) in MDA-MB-453 breast cancer cells, and explore the molecular mechanism of anti-angiogenesis in HER-2/neu-overexpressing breast cancer by genistein.
目的:观察染料木黄酮(genistein,Gen)对MDA-MB-453乳腺癌细胞血管内皮生长因子(vascularendothelialgrowthfactor,VEGF)表达的影响,探讨Gen抗HER-2/neu高表达乳腺癌血管生成的分子机制。
4) MDA-MB-231 cells
MDA-MB-231细胞
1.
Apoptosis and DNA damage in MDA-MB-231 cells induced by total synthetic Jaspine B;
全合成Jaspine B诱导人乳腺癌MDA-MB-231细胞凋亡及DNA损伤
2.
Methods: The inhibitory effect of different concentration of baicalin on the MDA-MB-231 cells was measured by MTT method.
方法:MTT法测定不同剂量黄芩苷对MDA-MB-231细胞株增殖的抑制作用;透射电镜检测细胞凋亡及黄芩苷的作用;流式细胞术分析黄芩苷对MDA-MB-231细胞周期变化的影响;RT-PCR技术分析黄芩苷对MDA-MB-231细胞bcl-2、baxmRNA水平表达的影响。
3.
Methods:MDA-MB-231 cells were cultured with As2O3 diflerint cokcenfrations.
方法:将不同浓度的As2O3与MDA-MB-231细胞共培养不同时间后,MTT比色法检测As2O3对MDA-MB-231细胞的生长抑制作用;TRAP-PAGE-银染法检测As2O3作用后细胞端粒酶活性的改变;RT-PCR法测定MDA-MB-231细胞hTERT-mRNA的表达。
5) MDA-MB-231
MDA-MB-231细胞
1.
Effect of 1,25-dihydroxyvitamin D_3 and Tamoxifen on cell cycle distribution and apoptosis of MDA-MB-231 cells transfected with ERα expression vector containing vitamin D response element;
1,25二羟维生素D_3联合他莫西芬对转染VDRE-Tk-ERα表达载体的MDA-MB-231细胞周期和凋亡的影响
2.
Objective To study the pro-apoptotic effects of 1,25 dihydroxyvitamin D3 in combination with Tamoxifen on MDA-MB-231 cells transfected with ERα expression vector containing VDRE elements and associated molecular mechanism.
方法将本室构建的含有4个拷贝维生素D反应元件(VDRE)和胸苷激酶启动子(Tk)的VDRE-Tk-ERα表达载体转染到MDA-MB-231细胞中,利用免疫荧光法检测1,25二羟维生素D3对雌激素受体α(ERα)的诱导表达并通过末端原位法检测1,25二羟维生素D3联合他莫西芬诱导转染该表达质粒的MDA-MB-231凋亡诱导效应。
6) MDA-MB-468 cell
MDA-MB-468细胞
1.
BACKGROUND & AIM:The inhibitory effect of arsenic trioxide(As2O3) on human breast cancer MDA-MB-468 cells and the change of syk expression.
背景与目的:探讨三氧化二砷(As2O3)对人乳腺癌MDA-MB-468细胞的生长抑制作用及其对syk表达的影响。
补充资料：MDA
孟烷二胺简称MDA，分子量170．3。透明液体，密度(23℃)0.91，粘度(25℃)19mPa·s。熔点-45℃，沸点107～126℃。
用作环氧树脂的固化剂，参考用量22phr，固化条件80℃／2h或130℃／30min。固化物热变形温度148～158℃。
用作环氧树脂的固化剂，参考用量22phr，固化条件80℃／2h或130℃／30min。固化物热变形温度148～158℃。
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
参考词条