1) disturbance of magnesium metabolism
镁代谢紊乱
2) Metabolic disorder
代谢紊乱
1.
Clinical analysis of the metabolic disorder in 66 cases of type 2 diabetes mellitus with obesity;
2型糖尿病肥胖患者66例代谢紊乱临床分析
2.
Characters of metabolic disorder in acute cerebral vascular disease and multivariate regress analysis of its related factors;
急性脑血管病的代谢紊乱特点及相关因素的多元回归分析
3.
Effects of Nanometer Propolis on Metabolic Disorder and Insulin Resistance in T2DM Rats;
纳米蜂胶对实验性2型糖尿病大鼠代谢紊乱与胰岛素抵抗的影响
3) Metabolism disorder
代谢紊乱
1.
The characteristics of metabolism disorder and the changes of serum homocysteine(tHcy) level in patients with obese type 2 diabetes mellitus;
2型糖尿病肥胖患者代谢紊乱特征及血清同型半胱氨酸水平的变化
4) metabolic disorders
代谢紊乱
1.
Objective To analyze the etiology, metabolic disorders and target organ damage of in-patients hypertensives whose blood pressure were difficult to control and to observe differences of metabolic disorders and target organ damage between essential hypertension(EH) and secondary hypertension(SH) in order to provide basis of correct diagnosis and therapy of these hyperensives.
目的了解住院的难以控制的高血压患者病因分布、合并的代谢紊乱及其靶器官损害的状况;并观察代谢紊乱、靶器官损害在原发性高血压和继发性高血压中分布的差异。
5) metabolic disturbance
代谢紊乱
1.
Objective To investigate the relationgship between the level of serum free fatty acids (FFA) and the metabolic disturbance of glucose and fat in type 2 diabetic mellitus (T2DM).
结论 2型糖尿病患者血清游离脂肪酸水平升高提示糖代谢紊乱的同时脂代谢亦异
2.
Conclusion The data suggestes that the changes of hemorheology is due to the metabolic disturbance.
结论 血液流变学改变与代谢紊乱有关。
6) lipid dysmetabolism
脂代谢紊乱
1.
Effects of lipid dysmetabolism in rats induced by high fat diet on glycometabolism and inflammatory factor;
高脂饲料喂养大鼠诱导的脂代谢紊乱对糖代谢及炎症因子的影响
2.
Study on effect of soybean protein on the lipids metabolism and inflammatory factors in lipid dysmetabolism rats;
大豆蛋白对脂代谢紊乱大鼠脂代谢及炎症因子作用的研究
补充资料:镁代谢紊乱
镁代谢紊乱 magnesium metabolism,disturbance of 镁摄入、排泄或体内过程障碍所致的疾病。镁是人体内位于钠、钾、钙之后最常见的阳离子。正常血清中镁为0.8~1.0毫摩/升,机体总镁量的一半存在于骨骼中 ,其余大部分存在于骨骼肌、心肌、肝、肾、脑等组织中,主要在细胞内,细胞外液仅占2%。 镁主要在小肠吸收 ,镁 能参与能 量贮存和蛋白质合成,在酶促反应中起辅酶作用,细胞外镁主要与神经肌肉传导和心血管张力有关,镁随尿、粪及汗液排泄。 血清镁低于 1.5 mEq /L为低镁血症 ,多见于长期禁食、腹泻、肾脏疾患时排镁过多、长期利用利尿药以及甲状腺功能减退等病,血清镁高于2.5mEq/L为高镁血症 ,多见于医源性给镁过多、肾功能衰竭等。缺镁时可致神经兴奋性增加,表现为反射亢进和震颤、手足徐动搐搦,亦可见嗜睡、惊厥等 ;高镁血症时可出现腱反射消失 、肌肉软瘫 、心动过缓,通常因呼吸肌麻痹而死亡。 镁代谢紊乱在临床上易与其他代谢紊乱混淆,治疗应针对原发病因。缺镁时可肌注硫酸镁。急性镁中毒时,可静注10%葡萄糖酸钙,因为钙对镁有拮抗作用。 |
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