1) Tyro-sine Kinase Protooncogene A (TrKA)
酪氨酸激酶原癌基因A
2) tyrosinase gene
酪氨酸酶基因
1.
A comparison of the sequences with known DNA sequences in human, cattle and mice showed that the region of tyrosinase gene is highly.
实验根据人、牛的酪氨酸酶(TYR,tyrosinase)基因序列设计了3对引物,扩增出猪的酪氨酸酶基因外显子2,3,4序列,其长度分别为210,135,182bp。
2.
The mutation of tyrosinase gene causes albimism.
酪氨酸酶是黑色素合成当中的关键酶，酪氨酸酶基因的突变将导致白化病。
3.
bisporus was used to construct the antisense expression vector of tyrosinase gene,and then it was transformed into A.
本研究克隆了蘑菇酪氨酸酶基因,构建了由蘑菇gpd启动子驱动的反义酪氨酸酶基因表达载体,采用农杆菌转化技术转化蘑菇AS2796,获得了大量的转化子。
3) Tyrosine kinase
酪氨酸激酶
1.
Effect of PDT on the activity of protein tyrosine kinase and phosphatase in H22 cells;
光动力疗法对H22细胞蛋白酪氨酸激酶及磷酸酶活性的影响
2.
Effects and mechanisms of epidermal growth factor receptor of tyrosine kinase on cancer;
表皮生长因子受体酪氨酸激酶抑制剂BPI-2009的抗肿瘤作用及其机制的研究
3.
Expression, purification and bioactivity determination of epidermal growth factor receptor protein tyrosine kinase;
表皮生长因子受体蛋白酪氨酸激酶的表达、纯化及活性测定
4) protein tyrosine kinase
酪氨酸激酶
1.
This paper reviewed recent progresses in the pharmacotherapeu- ties and clinical studies of the molecular-targeted anti-tumor drugs,including topoisomerase inhibitor, protein tyrosine kinase inhibitor,tumor angiogenesis inhibitor,PI3K-AKT-mTOR inhibitor and cyclin-de- pendent kinase inhib.
笔者综述了近年来DNA拓扑异构酶抑制剂、蛋白酪氨酸激酶抑制剂、肿瘤新生血管生成抑制剂、磷脂酰肌醇-3激酶/蛋白激酶B/哺乳动物雷帕霉素靶体(PI3K-AKT-mTOR)信号通路抑制剂、细胞周期依赖性蛋白激酶抑制剂的临床应用和临床研究进展。
5) Tyrosine kinase A
酪氨酸激酶A
1.
Objective To observe the changes of tyrosine kinase A(TrkA) in different cerebral regions of ovariectomized rats.
目的观察卵巢切除大鼠脑内不同部位酪氨酸激酶A(TrkA)表达的变化。
6) TrkA
酪氨酸激酶A
1.
Objective To investigate the expression of tyrosine kinase A(TrkA) in cerebral cortex neural stem cells of embryonic rats,so to find the functions of TrkA and demonstrate the possible mechanism of neural stem cells directional differentiation during the development of the nervous system.
目的通过研究不同发育阶段大鼠大脑皮层神经干细胞上TrkA(酪氨酸激酶A)表达的改变,为了解Tr-kA在神经系统发育中的作用与神经干细胞定向分化的可能机制打下基础。
2.
Objective To study the expressions of tyrosine kinase A (TrkA) and nerve growth factor (NGF) on cochlear corti in rats after whole cerebral ischemia-reperfusion.
目的探讨大鼠全脑缺血再灌注后酪氨酸激酶A(TrkA)和神经生长因子(NGF)在耳蜗Corti器的表达。
3.
The expression of NGF and TrkA mRNA/protein were measured with methods of RT-PCR and immunohistochemistry respectively at 2,4,6,12h and 24h after reperfusion in cerebral cortex ofrats.
目的探讨注射用血塞通对局灶性脑缺血再灌注神经生长因子(NGF)及酪氨酸激酶A(TrkA)含量变化的影响,并观察其对缺血性脑损伤的神经元保护作用。
补充资料：原癌基因
分子式：
CAS号：
性质：癌基因是正常的细胞基因，其表达产物与细胞正常生长、增殖和分化过程有关。但若被某种因素激活，就会转变成有转化细胞活力的癌基因而表达。由于这种表达在时空方面发生了混乱，能促使人或动物正常细胞发生转化和恶变。在人类细胞中此类癌基因称细胞癌基因或原癌基因。
CAS号：
性质：癌基因是正常的细胞基因，其表达产物与细胞正常生长、增殖和分化过程有关。但若被某种因素激活，就会转变成有转化细胞活力的癌基因而表达。由于这种表达在时空方面发生了混乱，能促使人或动物正常细胞发生转化和恶变。在人类细胞中此类癌基因称细胞癌基因或原癌基因。
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
参考词条