1) velopharygeal incompetence
腭咽关闭不全
1.
Objective: To explore a plastic surgical method to deal with velopharygeal incompetence after palatoplasty.
目的:探讨腭裂术后腭咽关闭不全的再手术治疗方法。
2) velopharyngeal incompetence
腭咽闭合不全
1.
Objective To explore a new concept of treating velopharyngeal incompetence.
目的 :应用自制的腭部牵张装置延长腭裂模型犬的腭部 ,探讨治疗腭咽闭合不全的新方法。
2.
OBJECTIVE To study the relationship between velopharyngeal morphology and velopharyngeal function in adult operated cleft palate patients with velopharyngeal incompetence ( VPI) .
<正>目的:分析腭裂术后腭咽闭合不全(VPI)患者成年期腭咽颈结构特征形态与腭咽功能之间的关系。
3.
We use it to lengthen the soft palate and correct velopharyngeal incompetence.
目的:探讨颊肌粘膜瓣的解剖及应用其延长软腭,同时改善腭咽闭合不全。
3) Velopharyngeal insufficiency
腭咽闭合不全
1.
Evaluation of outcomes of modified Furlow′s palatoplasty in treatment of velopharyngeal insufficiency using nasoendoscopy
应用鼻咽纤维镜评价改良Furlow's治疗腭咽闭合不全的临床效果
2.
Objective To study the sentential fluency of the patients with post-palatoplasty velopharyngeal insufficiency(VPI)for speech therapy of cleft palate.
本研究通过分析腭咽闭合不全患者的语句模式,探讨其内在规律,为临床语音治疗提供参考。
4) VPI
腭咽闭合不全
1.
ve To probe into clinical application value of S-A for velopharyngeal imcompetence (VPI) and find out related factors which affect curative effect.
目的 探讨S-A治疗腭咽闭合不全（VPI）的临床应用价值。
2.
The relationship between modified incision of cleft palate operation and post-operation velopharyngeal insufficiency(VPI);
目的 探讨腭裂手术切口设计的改良对术后腭咽闭合不全的影响。
5) RVPI
腭咽闭合不全率
1.
Rate of velopharyngeal incompetence(RVPI),F_1,F_2 and the F_1\'s energy of [i],the spike and fills of[zi]、[ci]、[si]、[ji]、[qi]、[xi]and Speech intelligibility were compared between anterior and posterior the operation,contrasted with 20 normal children.
比较手术前后腭咽闭合不全率(rate of velopharyngeal incompetence,RVPI),单元音[i]的F_2、F_3、F_1的能量值A_1,[zi]、[ci]、[si]、[ji]、[qi]、[xi]擦音乱纹(fills)及冲直条(strike)出现率及语音清晰度,并与健康儿童20例对照。
6) speech Aid
腭咽功能闭合不全
补充资料：二尖瓣关闭不全
二尖瓣关闭不全
mitral insufficiency
由于瓣增厚、变硬、弹性减弱或瓣卷曲、缩短、腱索增粗、钙化，使瓣在关闭时不能闭合，血流反流到左房，在舒张期过多的血液又流至左室，致使心室容量增加，负荷加重，引起左心室和左心房肥厚扩张。严重者出现左心衰竭，左室舒张终末压增高，左房压亦增高，产生肺淤血，肺动脉高压，最后引起右心室肥大、衰竭。临床主要表现为心悸、气短，听诊时可闻及心尖区全收缩期杂音，呈吹风样，第一心音减弱，肺动脉区第二音亢进，晚期可出现肝脾肿大、下肢浮肿等。
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
参考词条