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1)  brain hypoxia-ischemia injury
脑缺血缺氧性脑损伤
2)  hypoxic-ischemic brain damage
缺氧缺血脑损伤
1.
Objective To explore the protective effect of Lushi misture on hypoxic-ischemic brain damage neonatal rats.
目的探讨鹿石合剂对缺氧缺血脑损伤新生大鼠保护作用的机制。
2.
Methods Divided randomly the SD rat(n=64) of 7 days into 8 sets:4 different time false surgical operation sets and 4 corresponding hypoxic-ischemic brain damage sets(HIBD 6h,24h,72h,7d set),adopt Nogo-A polyclonal antibody immunohistochemistry SP methods.
结论Nogo-A在实验性缺氧缺血脑损伤后含量升高,可能抑制了中枢神经系统的再生。
3.
Methods Randomly divided the SD rat(n=64)of 7 days into 8 sets:4 different time hypoxic-ischemic brain damage sets(HIBD 6h,24h,72h,7d set)and 4 corresponding false surgical operation sets,using Ng-R polyclonal antibody immunohistochemistry SP methods to observe the Ng-R expression.
结论　Ng -R在实验性缺氧缺血脑损伤后含量升高,可能抑制了中枢神经系统的再生。
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3)  hypoxia-ischemia cerebral damage
缺血缺氧脑损伤
1.
Effects of Astragali polysaccharides on levels of calcium ion and excitatory amino acids for rats with hypoxia-ischemia cerebral damage;
黄芪多糖对缺血缺氧脑损伤大鼠脑组织Ca~(2+)和兴奋性氨基酸的影响
4)  hypoxic-ischemic brain damage
脑缺氧缺血损伤
1.
Cyclooxygenase 2 expression in neonatal rats contributes to hypoxic-ischemic brain damage;
新生大鼠脑缺氧缺血损伤时的COX-2表达变化
5)  Hypoxia ischemia brain damage
缺氧缺血脑损伤
1.
In the study the 7 d-day-old rats were used as hypoxia ischemia brain damage (HIBD) models.
用 7日龄 Wistar大鼠制成缺氧缺血脑损伤模型 ,从生后 7d到成年分 8个时间段对突触的质量进行了长期跟踪研究。
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6)  hypoxic-ischemic brain damage
缺氧缺血性脑损伤
1.
Expression of nerve growth inhibitor Nogo-A in neonatal rats with hypoxic-ischemic brain damage tissue and its significance;
Nogo-A蛋白在新生大鼠缺氧缺血性脑损伤的表达及意义
2.
Dynamic expression of glial cell line-derived neurotrophic factor and regulation mechanism of topiramate following hypoxic-ischemic brain damage in neonatal rats;
缺氧缺血性脑损伤新生大鼠胶质细胞源性神经营养因子表达及调控机制探讨
3.
Exogenous melatonin on TNF-α and IL-6 in serum of neonatal rats with hypoxic-ischemic brain damage;
褪黑素对新生大鼠缺氧缺血性脑损伤时血清TNF-α和IL-6的影响
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补充资料：闲脑
1.亦作"间脑"。 2.脑的一部分。在大脑两半球的中间﹐由许多形状不规则的灰质块和神经纤维构成。间脑包括丘脑和下丘脑。
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
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