1) hypoxic-ischemic brain damage
缺血缺氧性脑损伤
1.
Transforming growth factor-beta 1 and hypoxic-ischemic brain damage;
转化生长因子β_1与缺血缺氧性脑损伤
2.
Relationship between the HSP70 production and apoptosis in neonate rats after hypoxic-ischemic brain damage
新生大鼠缺血缺氧性脑损伤HSP70的合成与细胞凋亡的关系
3.
Objective: to observe, in different period, the morphological changes, the production of HSP70 and the formation of cell death of brain cells of neonate rats after undergoing hypoxic-ischemic brain damage(HIBD), to probe into the correlation between the post-HIBD HSP70 production and apoptosis, in order to put forward experimental and theoredical foundation for HIBD.
目的:利用新生大鼠缺血缺氧性脑损伤(hypoxic-ischemic brain damage HIBD)动物模型,观察缺血缺氧性脑损伤后不同时间脑细胞形态变化、HSP70的合成及细胞死亡形式,探讨HIBD后HSP70的合成与细胞凋亡的关系,为缺血缺氧性脑病提供实验和理论依据。
2) hypoxic Ischemic brain damage
缺血缺氧性脑损伤
1.
ALM To study the effects of ginkgo biloba extract (GbE) on expressions of neuron specific enolase (NSE) and S 100 protein(S 100) mRNA in newborn rat brain after hypoxic ischemic brain damage (HIBD) and the mechanism of GbE against HIBD.
目的 研究银杏叶提取物 (GbE)对新生鼠缺血缺氧性脑损伤 (HIBD)后脑组织神经元特异性烯醇化酶 (NSE)、S 10 0蛋白 (S 10 0 )mRNA表达的影响 ,以探讨GbE抗脑缺血缺氧的药理作用机制。
3) Hypoxic-ischemic brain damage(HIBD)
缺血缺氧性脑损伤
1.
[Background] Hypoxic-ischemic brain damage(HIBD) is one the major factors to make the newborn handicapped.
【背景】缺血缺氧性脑损伤(Hypoxic-ischemic brain damage)是临床上新生儿致残的最主要原因，其致死率致残率达47％，存活患儿中有1／3留有不同程度的神经系统后遗症。
2.
Objective: Hypoxic-ischemic brain damage(HIBD) could result in neonatal morbidity and mortality, some patients with HIBD developed into cerebral palsy and mental retardation.
目的 缺血缺氧性脑损伤(hypoxic-ischemic brain damage, HIBD)是严重威胁新生儿健康甚至致残的常见原因,目前临床上尚无较为理想的治疗措施。
4) brain hypoxia-ischemia injury
脑缺血缺氧性脑损伤
5) hypoxic-ischemic brain damage
缺氧缺血脑损伤
1.
Objective To explore the protective effect of Lushi misture on hypoxic-ischemic brain damage neonatal rats.
目的探讨鹿石合剂对缺氧缺血脑损伤新生大鼠保护作用的机制。
2.
Methods Divided randomly the SD rat(n=64) of 7 days into 8 sets:4 different time false surgical operation sets and 4 corresponding hypoxic-ischemic brain damage sets(HIBD 6h,24h,72h,7d set),adopt Nogo-A polyclonal antibody immunohistochemistry SP methods.
结论Nogo-A在实验性缺氧缺血脑损伤后含量升高,可能抑制了中枢神经系统的再生。
3.
Methods Randomly divided the SD rat(n=64)of 7 days into 8 sets:4 different time hypoxic-ischemic brain damage sets(HIBD 6h,24h,72h,7d set)and 4 corresponding false surgical operation sets,using Ng-R polyclonal antibody immunohistochemistry SP methods to observe the Ng-R expression.
结论 Ng -R在实验性缺氧缺血脑损伤后含量升高,可能抑制了中枢神经系统的再生。
6) hypoxia-ischemia cerebral damage
缺血缺氧脑损伤
1.
Effects of Astragali polysaccharides on levels of calcium ion and excitatory amino acids for rats with hypoxia-ischemia cerebral damage;
黄芪多糖对缺血缺氧脑损伤大鼠脑组织Ca~(2+)和兴奋性氨基酸的影响
补充资料：短暂性脑缺血发作
短暂性脑缺血发作
transient ischemic attack,TIA
急性脑血管病之一。指一时性脑缺血引起的一种局限性脑功能丧失，通常在24小时内完全缓解，不遗留重要神经功能缺陷。主要病因是脑动脉粥样硬化，亦可见于各种原因的动脉炎和心脏病。颈内动脉系统的脑缺血发作以病灶对侧的单瘫或偏瘫为常见，尤以上肢和面部为重，可伴有失语及精神症状。椎-基底动脉系统的脑缺血发作常见症状有眩晕、复视、构音障碍、吞咽困难、共济失调、单侧或双侧肢体瘫痪或感觉障碍等，至少两种以上症状共同出现。大脑后动脉供血不足可出现皮质盲，对侧同向偏盲。防治短暂性脑缺血发作，应针对每个人的病因，对发作次数多，考虑为微栓塞所致者，可慎重地选择抗凝治疗。主要病灶在颈部的动脉、颈内动脉颅内段或限于大脑中动脉主干者，可结合病人的具体情况考虑外科治疗。
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
参考词条