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1)  hypoxia-reoxygenation injury
缺氧再给氧损伤
1.
Study of Protective Effects of Catechins on Hypoxia-Reoxygenation Injury in Cultured Neonatal Rat Cardiac Myocytes and Their Mechanisms;
儿茶素对体外培养大鼠乳鼠心肌细胞缺氧再给氧损伤的保护作用及其机制研究
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2)  Anoxia/reoxygenation Injury
缺氧再给氧损伤
1.
AIM To study the effects of two hawthorn extracts (Vitexin-rhamnoside; Vitexin-2〃-O-glucoside) and hawthorn extracts preparation “AoShaEn Injection” on the adhesion between PMN (polymorphonuclear cell) and HUVEC (human umbilical vein endothelial cell) under anoxia/reoxygenation injury and its molecular mechanism.
方法以人脐静脉内皮细胞为实验对象,复制缺氧再给氧损伤模型,用酶标仪检测山楂叶提取物牡荆素鼠李糖甙、牡荆素葡萄糖甙及其制剂奥沙恩注射液对HUVEC缺氧60min复氧30min和复氧240min时PMN-HUVEC粘附的影响;应用流式细胞学方法,观察牡荆素鼠李糖甙、牡荆素葡萄糖甙及奥沙恩注射液对PMN表面粘附分子CD11/CD18表达的影响。
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3)  rehypoxicinjury
再缺氧损伤
4)  hypoxia-reoxygenation damage
缺氧再复氧损伤
1.
Protection of human renal tubular epithelial cells (HK-2 cells) from hypoxia-reoxygenation damage by recombinant adenovirus containing hCGPx gene;
重组hCGPx腺病毒对人肾小管上皮细胞(HK-2)缺氧再复氧损伤的保护作用
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5)  anoxia,reperfusion injury
缺氧再灌注损伤
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6)  hypoxia-reoxynation
缺氧再给氧
1.
Protection and its mechanism of catechin morphon on hypoxia-reoxynation induced injury in myocardial cells;
儿茶素单体对心肌细胞缺氧再给氧损伤的保护作用及机制研究
2.
Methods After establishing hypoxia-reoxynation model on myocardial cells,the content of LDH,MDA and apoptosis rate were examined.
方法在建立心肌细胞缺氧再给氧损伤模型基础上,研究川芎嗪对细胞培养液中乳酸脱氢酶及心肌细胞中丙二醛含量的影响;同时,探讨其对心肌细胞凋亡率的影响。
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补充资料：低氧性缺氧


低氧性缺氧
hypoxic hypoxia

由于空气中的氧稀薄、气道阻塞或弥散障碍，致肺泡进入毛细血管的氧量减少。
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
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