1) valvular heart disease
关闭不全
1.
Developments in percutaneous coronary intervention technical transcatheter therapy in valvular heart disease have become a focal point of research.
随着介入技术的普及,心脏瓣膜病介入治疗成为介入心脏病学的新热点,瓣膜狭窄的介入治疗已经基本成形,目前关于瓣膜关闭不全的介入治疗出现新的巨大进步,现就瓣膜病的介入治疗及其发展做一综述,已期引起医生高度关注。
2) inadequate
关闭不全的
3) Mitral regurgitation
二尖瓣关闭不全
1.
Surgical treatment of 27 cases of coronary artery disease with ischemic mitral regurgitation;
冠心病伴缺血性二尖瓣关闭不全27例手术治疗分析
2.
Analysis on the causes of mitral regurgitation after percutaneous transvenous mitral valvuloplasty;
经皮二尖瓣球囊成形后二尖瓣关闭不全及其原因分析
3.
Follow-up observation of mitral valve repair in treatment of mitral regurgitation with depressed left ventricular function;
二尖瓣修复术治疗左室功能减退的二尖瓣关闭不全患者效果的随访观察
4) mitral insufficiency
二尖瓣关闭不全
1.
Fifty-three cases of valve repair for mitral insufficiency;
53例二尖瓣关闭不全的外科修复
2.
Method: To select 60 valval disease patients diagnosed by Colour Ultrasound Doppler Flowmeter and Magnetic Resonance Imaging (MRI) detections, among those 30 patients suffering from mitral insufficiency and mitral stenosis, 30 patients arotic insufficiency and aortic stenosis, being engaged in CADL or PADL detection for them.
方法 :选择经彩色超声多普勒、MRI检查确诊的瓣膜病患者 6 0例 ,其中二尖瓣关闭不全、二尖瓣狭窄的患者 30例 ;主动脉瓣关闭不全、主动脉瓣狭窄的患者 30例 ,对他们进行CADL或PADL的检测。
3.
Objective To summarize and analyze the clinical effect of 34 mitral insufficiency treated with mitral valvuloplasty.
目的总结34例行二尖瓣修复成形术的二尖瓣关闭不全患者的临床疗效。
5) mitral valve regurgitation
二尖瓣关闭不全
6) Tricuspid insufficiency
三尖瓣关闭不全
1.
Significance of Tei index for evaluating right heart function in tricuspid insufficiency;
Tei指数评价三尖瓣关闭不全时右心功能改变及其意义
2.
Objective To discuss the influence of cardiovalvulotransplantation under extracorporeal circulation on tricuspid insufficiency secondary to rheumatic heart disease(TR).
目的探讨风心病继发三尖瓣关闭不全(TR)对体外循环瓣膜置换术效果的影响及手术适应证。
参考词条
三尖瓣重度关闭不全
缺血性二尖瓣关闭不全
功能性三尖瓣关闭不全
三尖瓣膜关闭不全
心脏瓣膜关闭不全
非缺血性二尖瓣关闭不全
主动脉瓣关闭不全(Aortic valveinsuff iciency)
主动脉瓣关闭不全/超声检查
二尖瓣关闭不全/外科手术
甲基丙烯酸丁酯
补充资料:二尖瓣关闭不全
二尖瓣关闭不全
mitral insufficiency
由于瓣增厚、变硬、弹性减弱或瓣卷曲、缩短、腱索增粗、钙化,使瓣在关闭时不能闭合,血流反流到左房,在舒张期过多的血液又流至左室,致使心室容量增加,负荷加重,引起左心室和左心房肥厚扩张。严重者出现左心衰竭,左室舒张终末压增高,左房压亦增高,产生肺淤血,肺动脉高压,最后引起右心室肥大、衰竭。临床主要表现为心悸、气短,听诊时可闻及心尖区全收缩期杂音,呈吹风样,第一心音减弱,肺动脉区第二音亢进,晚期可出现肝脾肿大、下肢浮肿等。
说明:补充资料仅用于学习参考,请勿用于其它任何用途。