裂原激活蛋白激酶,MAPK,在线英语词典,英文翻译,专业英语

1) MAPK

裂原激活蛋白激酶

43, contains a mitogen-activated protein kinase (MAPK) domain with a dual-phosphorylation activation motif TDY (Thr-Asp-Tyr) and a C-terminus domain that feature a typical MAPK belonging to subgroup V.

裂原激活蛋白激酶(MAPK)级联系统负责把接受自胞外或胞内的信号进一步传递和放大而最终作用于特异的转录因子,从而启动或调控基因的表达。

2) mitogen-activated protein kinase

丝裂原激活蛋白激酶

Abstract: Objective To understand the role of p38 mitogen-activated protein kinase (p38MAPK) in the expression of in-ducible nitric oxide synthase (iNOS) and NO production in human endothelial cells under the stimulation by lipopolysaccha-ride (LPS).

目的研究p38丝裂原激活蛋白激酶(p38 MAPK)在脂多糖(LPS)诱导的人内皮细胞－ECV304诱导型一氧化氮合酶(iNOS)表达和一氧化氮(NO)产生中的作用。

3) p38 mitogen-activated protein kinase

p38丝裂原激活蛋白激酶

Objective:To identify the role of p38 mitogen-activated protein kinase(p38 MAPK)and its specific inhibitor,SB203580,in airway inflammation and mucus hypersecretion in vivo.

目的探讨小鼠吸入丙烯醛后是否出现气道黏液高分泌及其产生机制,观察p38丝裂原激活蛋白激酶(p38MAPK)信号通路在气道炎症损伤、黏液高分泌中的作用,以及应用p38MAPK特异性抑制剂SB203580干预后对气道炎症、MUC5AC的影响。

Objective: To investigate the effects of pentoxifylline (PTX) on activation of p38 Mitogen-activated protein kinase, and to study the protective mechanism of PTX on endothel cell monolayer permeability.

目的:观察己酮可可碱(PTX)对p38丝裂原激活蛋白激酶(MAPK)激活的影响,探讨PTX保护内皮细胞单层通透性的机制。

Though there was one literature about p38 mitogen-activated protein kinase(MAPK) involed in regulation of LPS-induceed ICAM- 1 protein.

尽管已有文献报道p38丝裂原激活蛋白激酶（MAPK）可能参与LPS诱导内皮细胞ICAM-1蛋白的表达，但仍不清楚p38MAPK作用的位点是在ICAM-1蛋白翻译水平或是在基因转录水平。

4) MAPK

丝裂原激活蛋白激酶

THE ROLE OF MAPK IN LPS-INDUCED iNOS EXPRESSION IN RAT SCHWANN CELLS;

丝裂原激活蛋白激酶在内毒素脂多糖诱导大鼠施万细胞一氧化氮合酶表达中的作用

BACKGROUND AND AIM:To investigate the effects of thioglycolic acid on the activities of MAPK and MPF protein kinases during progesterone-induced Xenopus oocyte maturation process in vitro.

背景与目的:探讨巯基乙酸(TGA)对孕酮诱导爪蟾卵母细胞体外成熟过程中丝裂原激活蛋白激酶(MAPK)和成熟促进因子(MPF)蛋白激酶活性的影响。

5) Mitogen-activated protein kinases

丝裂原激活蛋白激酶类

6) p38MAPK

p38丝裂原激活蛋白激酶

OBJECTIVE To investigate the effect of astilbin on activation of p38 mitogen activated protein kinase(p38MAPK) in activated T cells of mouse heart transplantation model with acute rejection.

目的:用小鼠心脏移植急性排斥反应体外模型,探讨落新妇苷对心脏移植排斥反应中活化T细胞p38丝裂原激活蛋白激酶(p38MAPK)信号传导通路的影响。

This article was presented to study the effects of tetrandrine(Tet) on phenotypic modulation of vascular smooth muscle cells(VSMC) and p38 mitogen-activated protein kinase(p38MAPK) as well as mitogen-activated protein kinase phosphotase-1(MKP1) after vascular intimal injury.

研究粉防己碱(tetrandrine,Tet)抑制内膜损伤后血管平滑肌细胞(VSMC)表型转化和对p38丝裂原激活蛋白激酶(p38MAPK)及丝裂原激活蛋白激酶磷酸酶-1(MKP-1)表达的影响。

参考词条

补充资料：乙酰化纤溶酶原-链激酶激活剂复合物

药物名称：乙酰化纤溶酶原-链激酶激活剂复合物

英文名：APSAC

该药对血块有选择性高亲和力且半衰期长，清除半衰期为70分钟，故纤维蛋白溶解作用缓慢而持久。用药后5-8小时仍可维持有效溶栓血药浓度，有报告作用维持7-10天，而SK用药后3小时血中即完全清除，因此可视为长效SK。

APSAC用药特点是短RV间内一次给予全部剂量。据报告冠状动脉内溶栓5分钟内注射5-30mg，1-4小时后闭塞动脉再通率为70-83％。静脉给药5分钟内注入5-30mg，再通率为60-100％。该药全身纤溶作用微弱，用药简单，药物作用时间长，然而再闭塞率可达20％，但平均再闭塞率仍较SK、rt-PA为低。

类别：溶栓药

说明：补充资料仅用于学习参考，请勿用于其它任何用途。

首页 \| 翻译 \| 影视 \| 论坛 \| 索引 p38分裂原激活蛋白激酶 p38-分裂原激活蛋白激酶

1) MAPK 裂原激活蛋白激酶 1. 43, contains a mitogen-activated protein kinase (MAPK) domain with a dual-phosphorylation activation motif TDY (Thr-Asp-Tyr) and a C-terminus domain that feature a typical MAPK belonging to subgroup V. 裂原激活蛋白激酶(MAPK)级联系统负责把接受自胞外或胞内的信号进一步传递和放大而最终作用于特异的转录因子,从而启动或调控基因的表达。 2) mitogen-activated protein kinase 丝裂原激活蛋白激酶 1. Abstract: Objective To understand the role of p38 mitogen-activated protein kinase (p38MAPK) in the expression of in-ducible nitric oxide synthase (iNOS) and NO production in human endothelial cells under the stimulation by lipopolysaccha-ride (LPS). 目的研究p38丝裂原激活蛋白激酶(p38 MAPK)在脂多糖(LPS)诱导的人内皮细胞－ECV304诱导型一氧化氮合酶(iNOS)表达和一氧化氮(NO)产生中的作用。 3) p38 mitogen-activated protein kinase p38丝裂原激活蛋白激酶 1. Objective:To identify the role of p38 mitogen-activated protein kinase(p38 MAPK)and its specific inhibitor,SB203580,in airway inflammation and mucus hypersecretion in vivo. 目的探讨小鼠吸入丙烯醛后是否出现气道黏液高分泌及其产生机制,观察p38丝裂原激活蛋白激酶(p38MAPK)信号通路在气道炎症损伤、黏液高分泌中的作用,以及应用p38MAPK特异性抑制剂SB203580干预后对气道炎症、MUC5AC的影响。 2. Objective: To investigate the effects of pentoxifylline (PTX) on activation of p38 Mitogen-activated protein kinase, and to study the protective mechanism of PTX on endothel cell monolayer permeability. 目的:观察己酮可可碱(PTX)对p38丝裂原激活蛋白激酶(MAPK)激活的影响,探讨PTX保护内皮细胞单层通透性的机制。 3. Though there was one literature about p38 mitogen-activated protein kinase(MAPK) involed in regulation of LPS-induceed ICAM- 1 protein. 尽管已有文献报道p38丝裂原激活蛋白激酶（MAPK）可能参与LPS诱导内皮细胞ICAM-1蛋白的表达，但仍不清楚p38MAPK作用的位点是在ICAM-1蛋白翻译水平或是在基因转录水平。 4) MAPK 丝裂原激活蛋白激酶 1. THE ROLE OF MAPK IN LPS-INDUCED iNOS EXPRESSION IN RAT SCHWANN CELLS; 丝裂原激活蛋白激酶在内毒素脂多糖诱导大鼠施万细胞一氧化氮合酶表达中的作用 2. BACKGROUND AND AIM:To investigate the effects of thioglycolic acid on the activities of MAPK and MPF protein kinases during progesterone-induced Xenopus oocyte maturation process in vitro. 背景与目的:探讨巯基乙酸(TGA)对孕酮诱导爪蟾卵母细胞体外成熟过程中丝裂原激活蛋白激酶(MAPK)和成熟促进因子(MPF)蛋白激酶活性的影响。 5) Mitogen-activated protein kinases 丝裂原激活蛋白激酶类 6) p38MAPK p38丝裂原激活蛋白激酶 1. OBJECTIVE To investigate the effect of astilbin on activation of p38 mitogen activated protein kinase(p38MAPK) in activated T cells of mouse heart transplantation model with acute rejection. 目的:用小鼠心脏移植急性排斥反应体外模型,探讨落新妇苷对心脏移植排斥反应中活化T细胞p38丝裂原激活蛋白激酶(p38MAPK)信号传导通路的影响。 2. This article was presented to study the effects of tetrandrine(Tet) on phenotypic modulation of vascular smooth muscle cells(VSMC) and p38 mitogen-activated protein kinase(p38MAPK) as well as mitogen-activated protein kinase phosphotase-1(MKP1) after vascular intimal injury. 研究粉防己碱(tetrandrine,Tet)抑制内膜损伤后血管平滑肌细胞(VSMC)表型转化和对p38丝裂原激活蛋白激酶(p38MAPK)及丝裂原激活蛋白激酶磷酸酶-1(MKP-1)表达的影响。参考词条丝裂原激活蛋白激酶（MAPK）丝裂原激活蛋白激酶p38 p38 丝裂原激活蛋白激酶分裂原激活蛋白激酶 p38丝裂原激活蛋白激酶抑制剂有丝分裂原激活蛋白激酶丝裂原激活蛋白激酶类/代谢丝裂原激活蛋白激酶阻断剂丝裂原激活蛋白激酶磷酸酶-1 有丝分裂原激活蛋白激酶p38 p38丝裂原激活蛋白激酶类促分裂原激活蛋白激酶（MAPK）有丝分裂原激活蛋白激酶类 p38丝裂原激活蛋白激酶（p38 MAPK）丝裂原激活蛋白激酶阻断剂PD98059 复合显微结构参数预算外收支补充资料：乙酰化纤溶酶原-链激酶激活剂复合物药物名称：乙酰化纤溶酶原-链激酶激活剂复合物英文名：APSAC 该药对血块有选择性高亲和力且半衰期长，清除半衰期为70分钟，故纤维蛋白溶解作用缓慢而持久。用药后5-8小时仍可维持有效溶栓血药浓度，有报告作用维持7-10天，而SK用药后3小时血中即完全清除，因此可视为长效SK。 APSAC用药特点是短RV间内一次给予全部剂量。据报告冠状动脉内溶栓5分钟内注射5-30mg，1-4小时后闭塞动脉再通率为70-83％。静脉给药5分钟内注入5-30mg，再通率为60-100％。该药全身纤溶作用微弱，用药简单，药物作用时间长，然而再闭塞率可达20％，但平均再闭塞率仍较SK、rt-PA为低。类别：溶栓药说明：补充资料仅用于学习参考，请勿用于其它任何用途。
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