1) renal fibrosis
肾脏纤维化
1.
Relation of renal tissue aldosterone and its receptor with renal fibrosis in spontaneously hypertensive rats;
自发性高血压大鼠肾组织醛固酮及其受体与肾脏纤维化
2.
Effect of benazepril on renal fibrosis in spontaneously hypertensive rats;
盐酸贝那普利对自发性高血压大鼠肾脏纤维化的影响
3.
Effects of enalapril and valsartan on renal fibrosis of chronic kidney failure rats;
依那普利联合缬沙坦对慢性肾功能衰竭大鼠肾脏纤维化的影响
2) renal interstitial fibroblasts
肾脏成纤维细胞
1.
Effects of rosiglitazone on expression of transforming growth factor β1 and fibronectin in renal interstitial fibroblasts cultured with cyclosporine A;
罗格列酮对环孢素A作用下大鼠肾脏成纤维细胞转化生长因子-β1和层黏连蛋白表达的影响
3) Renal fibrosis
肾纤维化
1.
Effects of oxymatrine on expression of nuclear factor kappa B in kidneys of rats with adriamycin-induced chronic renal fibrosis;
氧化苦参碱对阿霉素大鼠慢性肾纤维化组织中核因子-κB表达的影响
2.
Effects of Broad-Spectrum Matrix Metalloproteinases Inhibitor on Hypertensive Renal Fibrosis in Stroke-prone Spontaneously Hypertensive Rats;
广谱基质金属蛋白酶抑制剂对高血压肾纤维化的影响
3.
Relationship between transforming growth factor-β_1 signal transduction and renal fibrosis of diabetes;
糖尿病肾病中转化生长因子-β_1信号转导与肾纤维化
4) kidney fibrosis
肾纤维化
1.
Inhibitory mechanism of Ginkgo biloba extract,Captopril,and Vaisartan on kidney fibrosis of diabetic nephropathy rats and their comparison;
银杏叶提取物、卡托普利和缬沙坦抑制糖尿病肾病大鼠肾纤维化的机制及其比较
2.
Objective To explore the activin A expression in rat experimental kidney fibrosis induced by UUO and analyze its role in the pathogenesis of kidney fibrosis.
目的:研究激活素A(ACT A)在大鼠实验性肾纤维化中的表达,分析其在肾纤维化中的作用;并利用外源卵泡抑素(FS)干预实验进一步证明ACT A参与了肾小管纤维化发生这一论断。
5) cardiac fibrosis
心脏纤维化
1.
Effects of Myofibroblasts on the Cardiac Fibrosis of the Infarcted Rat Heart;
肌成纤维细胞在大鼠急性心肌梗死后心脏纤维化中作用的研究
2.
Purpose To observe whether the leptin receptor mRNA is expressed and cardiac fibrosis is present in hearts of Streptozotocin - diabetic rats.
结果 STZ糖尿病大鼠和对照鼠心肌均可转录表达短型瘦素受体mRNA ,两组之间无显著差别 ,但糖尿病心肌成纤维细胞短型瘦素受体mRNA表达较高 ,伴心功能减退和显著的心脏纤维化病理改变。
3.
Cardiac pressure overload stimulates cardiovascular hypertrophy and cardiac fibrosis, often leading to heart failure.
我们发现，TLR激动剂及BCG在没有明显改变血流动力学前提下，显著减轻压力过负荷所致心血管肥厚和心脏纤维化；TLR4拮抗剂能够逆转TLR4激动剂以及BCG引起的抗心血管肥厚和心脏纤维化的作用。
6) hepatic fibrosis
肝脏纤维化
1.
Expression of AT_(1a)mRNA in rat hepatic stellate cells and the study on mechanism of hepatic fibrosis;
AT_(1a)mRNA在大鼠肝脏星状细胞中的表达及对肝脏纤维化作用机制的研究
补充资料：碳(石墨)纤维增强体(见碳纤维、石墨纤维)
碳(石墨)纤维增强体(见碳纤维、石墨纤维)
earbon(graPhite)fibrereinforeements
，、、里z耽维增强体earbon(graphi现inforcements用于复合材料中承受负荷，田的磷纤始知五史纤俯亡e)fibre起增强作
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
参考词条