2) Inducible nitric oxide synthase
诱导型一氧化氮合酶
1.
Effects of inducible nitric oxide synthase derived nitric oxide on apoptosis of HL-60 cells co-cultured with RAW264.7 macrophages;
巨噬细胞中诱导型一氧化氮合酶来源的NO对共培养HL-60细胞凋亡的影响
2.
Inhibitive effect of tacrolimus on expression of inducible nitric oxide synthase in rats following contusion spinal cord injury;
FK506抑制脊髓挫伤后诱导型一氧化氮合酶表达的实验研究
3.
Effect of melatonin on expression of inducible nitric oxide synthase in human retinal pigment epithelial cells cultured with high glucose;
褪黑素对高糖刺激人视网膜色素上皮细胞诱导型一氧化氮合酶表达的影响
3) Inducible nitric oxide synthase(iNOS)
诱导型一氧化氮合酶
1.
To probe the activity of inducible nitric oxide synthase(iNOS) in liver,the concentration of nitric oxide(NO) in serum and its inhibition or killness on coccidia in Eimeria stiedai infected rabbits,the infected rabbits were injected with the substrate of nitric oxide synthase L-arginine(L-Arg) and the inhibitor of inducible nitric oxide synthase L-aminoguanidine(L-AG) through the abdominal cavity.
stiedai)感染兔肝脏诱导型一氧化氮合酶(iNOS)活性和血清NO浓度变化以及NO对兔球虫的抑制或杀伤作用,以腹腔注射途径给予球虫感染家兔一氧化氮合酶(NOS)底物L-精氨酸(L-Arg)和iNOS抑制剂L-氨基胍(L-AG),对家兔球虫感染时肝脏iNOS活性、血清NO浓度、每克粪便的球虫卵囊数(OPG)、肝的剖检和镜下病变及其病变计分进行了研究。
2.
Object: To investigate the different expression of inducible nitric oxide synthase(iNOS) among normal buccal mucous (NBM), oral submucous fibrosis(OSF), oral lichen planus(OLP), oral leukoplakia(OLK) and oral squamous cell carcinoma(OSCC), so as to evaluate the carcinogenic ability of OSF and discuss the role of iNOS in the carcinogenesis of OSF.
目的通过研究诱导型一氧化氮合酶(inducible nitric oxidesynthase,iNOS)在正常口腔粘膜(Normal buccal mucous,NBM)、口腔粘膜下纤维化(oral submucous fibrosis,OSF)、口腔扁平苔藓(orallichen planus,OLP)、口腔白斑(oral leukoplakia,OLK)和口腔鳞癌(oral squamous cell carcinoma,OSCC)组织中的表达差异,探讨OSF癌变潜能大小及iNOS在其癌变过程中的可能作用。
4) iNOS
诱导型一氧化氮合酶
1.
To construct the the rAAV vectot delivery of iNOS mRNA antisense;
诱导型一氧化氮合酶反义RNA重组腺相关病毒载体的构建
2.
Expression and Significance of HIF-1α and iNOS in human middle ear cholesteatoma epithelium;
缺氧诱导因子-1α及诱导型一氧化氮合酶在中耳胆脂瘤上皮中的表达及意义
3.
iNOS,ONOO~- expression indued by alumina and ultra-high-molecular-weight polyethylene wear particles;
氧化铝陶瓷和高分子聚乙烯磨损颗粒刺激诱导型一氧化氮合酶和过氧亚硝基阴离子生成的实验研究
5) Induced nitric oxide synthase
诱导型一氧化氮合酶
1.
Objective To investigate the role of induced nitric oxide synthase and nitric oxide and of hyperbaric oxygen therapy on the hepatic artery ligation rat.
目的探讨大鼠肝脏及血清诱导型一氧化氮合酶、一氧化氮在肝动脉结扎损伤中的意义及高压氧的治疗作用。
6) iNOS
诱导型一氧化氮合成酶
1.
Expression and significance of iNOS in rat corneal neovascularization;
诱导型一氧化氮合成酶在大鼠角膜新生血管模型的表达及意义
2.
Methods Capillary electrophoresis and reverse transcription-polymerase chain reaction (RT-PCR) were used to measure the mRNA expressions of albumin, TNFα , IL-1α, IL-6R and iNOS respectively and the change of plasma albumin on 1,3,6 and 9 postburn day (PBD) in rats in early enteral feeding group (EF) and in L-arginine enriched e.
方法 应用反转录聚合酶链式反应 (RT -PCR)和毛细管电泳半定量检测白蛋白等的mRNA表达 ,观察富含精氨酸早期肠内营养组 (AEF组 )及单纯早期肠内营养组(EF)大鼠在烧伤后第 1、3、6、9天肝脏白蛋白、肿瘤坏死因子α(TNFα)、白细胞介素 1α(IL - 1α)、白细胞介素 6受体 (IL - 6R)、诱导型一氧化氮合成酶 (iNOS)的mRNA表达及血浆白蛋白的变化。
3.
Objective To investigate the effects of UVA irradiation on expression of inducible nitric oxide synthase(iNOS) mRNA,iNOS protein and the change of morphology of Human primary fibroblasts.
目的通过检测UVA照射后人皮肤成纤维细胞的细胞形态及诱导型一氧化氮合成酶(iNOS)和一氧化氮(NO)表达,初步探讨UVA引起成纤维细胞光损伤的发病机制。
补充资料:光诱导型基因传递系统
东京大学(the university of tokyo)研究者的最新报道指出,他们解决了在基因治疗中的最大挑战—将dna安全、有效地转入细胞。他们开发了一种光诱导的基因传递系统(gene delivery system):将dna分子和光敏感化合物组装进一个简单的结构中,进而使实验操作对细胞的毒害降低到最小。
类似dna的大分子进入细胞的主要方法是通过胞吞作用(endocytosis),细胞外膜内陷将化合物单独隔离在一个称作内涵体(endosome)的区室中。光敏分子(photosensitive molecule)在接受光激发的时候帮助dna分子破膜而出。在dna周围包被上这种分子,使得研究者对细胞内其他细胞器和膜结构的破坏降低到最小。
说明:补充资料仅用于学习参考,请勿用于其它任何用途。