1.
The Effects of Bradykinin on Blood Brain Barrier in Ischemic Area after Focal Ischemia in Rats;
缓激肽对大鼠缺血区血脑屏障的影响
2.
The QSAR research of bradykinin-potentiating pentapeptides
血管舒缓激肽促进剂的QSAR研究
3.
BDKRB1 and cervical carcinoma occurrence and development
缓激肽B1(BDKRB1)与宫颈癌的发生与发展
4.
Moreover, the enzyme hydrolyzed bovine low molecular mass kininogen and releasing bradykinin.
另外,此酶能够水解牛低分子量的激肽原,释放舒缓激肽。
5.
The Function of Bradykinin Receptors in Cerebral Ischemia-reperfusion;
缓激肽受体在脑缺血再灌流中的作用研究
6.
In Vitro Study of the Mechanism of Bradykinin Selective Opening the Blood Brain Barrier;
缓激肽选择性开放血脑屏障的体外探讨
7.
Effects of bradykinin on the sodium channel of dorsal root ganglion neurons;
缓激肽对背根节神经元钠通道电流的作用
8.
Mechanism of Bradykinin Selectively Increasing the Permeability of Blood-tumor Barrier;
缓激肽选择性开放血肿瘤屏障机制的研究
9.
Y-27632 inhibits the opening of blood tumor barrier caused by bradykinin
Y-27632抑制缓激肽选择开放血肿瘤屏障的研究
10.
Inhibition of bradykinin to PDGF-induced mesangial cells proliferation
缓激肽对PDGF诱导的肾小球系膜细胞增殖的影响
11.
In Vitro Effect of Low Dose Bradykinin to Calcium-activated Potassium Currents on C6 Blood-brain Tumor Barrier of Rats;
缓激肽对离体的C6大鼠血脑肿瘤屏障钙激活钾电流的作用
12.
Relationship between Gene Polymorphism of Bradykmin Receptor β_2 (BKβ_2R) and Essential Hypertension
缓激肽β_2受体基因多态性与原发性高血压关系的研究现状
13.
Effects of Bradykinin on the Expression of pCREB, Interleukin-1β and Interleukin-6 in C6 Glioma Cells;
缓激肽对胶质瘤细胞磷酸化CREB、IL-1β和IL-6表达的影响
14.
Mediation of Tumor Necrosis Factor-α to Bradykinin in Opening the Blood-brain Barrier;
肿瘤坏死因子-α介导缓激肽开放血脑屏障的作用
15.
The Protective Effects of Bradykinin Preconditioning and Brain Ischemic Preconditioning on Cerebral Ischemia;
缓激肽预处理及脑缺血预处理对脑缺血保护作用的研究
16.
The Role of Glioma Cell in Bradykinin Selective Modulation of the Blood Tumor Barrier;
胶质瘤细胞在缓激肽选择性开放血肿瘤屏障中的作用探讨
17.
Relationship between Bradykinin B_1 Receptor 1098A/G Gene Polymorphism and Antihypertensive Therapy Effect;
缓激肽B_1受体1098A/G多态性对ACEI类降压药物疗效的影响
18.
COX-2 and HO-1 Are Involved in the Delayed Preconditioning Elicited by Bradykinin in Rat Hearts;
COX-2和HO-1参与缓激肽诱发的晚期心肌保护作用