1.
Construction and Identification of Bait Expression Plasmid of Co-activator CBP
协同激活分子CBP诱饵表达质粒的构建和鉴定
2.
Human Papillomavirus 16 E7-Specific Cytotoxic Lymphocyte Induction Through Mouse B7-1 Costimulating with E7 of HPV16 or E7C Subgene Fragment;
人乳头瘤病毒16型E7或E7C与共激活分子B7-1协同诱导特异性CTL的研究
3.
CD28 Antibody and MHC Class Ⅱ Expressing Thyrocytes Stimulate T Lymphocyte Proliferation and Activation in Vitro
CD28抗体协同表达MHC Ⅱ类分子的甲状腺细胞刺激自身T细胞增殖和活化
4.
CD167 Acts as a Novel Costimulatory Receptor in T Cell Activation;
CD167：一类新的T淋巴细胞协同刺激分子受体
5.
Expression of Endometrial Cancer p72,AIB1,erbB-2 Detected by Real-time Quantitative RT-PCR and Coactivation to ERα;
子宫内膜癌p72、AIB1、erbB-2的表达及其对雌激素受体活性的协同作用
6.
The Role of Cytokines and Costimulation on Herpetic Stromal Keratitis in a Murine Model;
细胞因子与协同刺激分子对鼠单纯疱疹性角膜基质炎的介导作用
7.
Synergistic Activation of Eukaryotic Gene Transcription by Multiple Upstream Sites
真核基因转录激活的多位点协同调控
8.
Studies of the Functions of the Transmembrane Adapter Protein Cbp in Receptor Tyrosine Kinases Related Signal Transduction and Cell Activities;
跨膜接头蛋白Cbp在受体型酪氨酸激酶相关信号转导和细胞活动中的功能研究
9.
The Roles of Costimulatory Molecules CD80 and CD86 in Experimental Allergic Neuritis;
协同刺激分子CD80和CD86在实验性变态反应性神经炎发病中的作用
10.
Experimental Study on Tumor Immunity of Hepatocarcinoma Cells Induced by IL-12、IFN-γ Synergizes B7-1 Costimulation;
IL-12、IFN-γ协同共刺激分子B7-1诱导肝癌细胞肿瘤免疫的实验研究
11.
Effect of Blockage of Costimulatory Signal CD86 on Murine Abortion-prone Model;
阻断CD86协同刺激分子对自然流产模型孕鼠的影响
12.
Role of T Lymphocyte-associated Costimulatory Molecules in the Pathogenesis of Severe Pneumonia
T淋巴细胞相关协同刺激分子在重症肺炎发病机制中的作用研究
13.
Research on the Expression and Clinical Significance of Synergistic Co-stimulatory Molecules B7-H3 in Cervical Cancer
协同共刺激分子B7-H3在宫颈癌中表达及其临床意义的研究
14.
New Findings about the Regulation of Costimulatory Molecules in Maternal-fetal Immunotolerance
重视协同刺激分子在母胎免疫耐受中的调控作用
15.
molecular laser isotope separation
分子激光同位素分离
16.
Study on the correlation of costimulatroy molecule B7 and transforming growth factor-betal in pathogenesis of endometriosis;
协同刺激分子B7、转化生长因子TGF-β1在子宫内膜异位症发病机制的相关性研究
17.
HSV-1 Immediate Early Protein ICP0 Ubiquitinates TRAF6 and Synergies Its Activation of NF-κB
HSV-1早早期蛋白ICP0泛素化TRAF6并协同其激活NF-κB
18.
The Impact of K-ras~(G12D) Activation and p53 Loss upon Tumorigenesis
p53缺失协同K-ras~(G12D)激活对细胞成瘤性的影响