1.
The Effects of Perindopril or Fosinopril Therapy on Congestive Heart Failure and Proinflammatory Cytokines;
培哚普利、福辛普利对心衰细胞因子的影响比较
2.
Study of Cadiocyte Apoptosis、Cytokine and B-type Natriuretic Peptide Effected by Kangdaxin in Heart Failure Rats
康达心对慢性心衰鼠心肌细胞凋亡、细胞因子及B-型利钠肽的影响
3.
Simulation Study of Heart Failure Based on Heart Cell Model;
基于心肌细胞模型的心力衰竭仿真研究
4.
Preliminary exploration for a method of analyzing gene expression profiles in myocardium cells of heart failure patients
人心力衰竭心肌细胞基因表达谱分析方法初探
5.
Losartan reduces cardiomyocyte apoptosis in rats with heart failure
氯沙坦降低心力衰竭大鼠心肌细胞凋亡
6.
Study of Cyclic Amp in Cardiomyocytes of Heart Failure Rat which Transfected Ad5-ADRbeta2-EGFP by Adenovirus Vector
重组腺病毒转染心衰大鼠心肌细胞cAMP水平变化
7.
Cellular Basis of Abnormal Calcium Cycling of Failing Rabbits Myocytes and Effects of Metoprolol and Losartan;
家兔心衰心肌细胞钙循环异常的细胞学基础及药物干预研究
8.
An Experimental Study on the Effect on the Cardiac Cells After Self Bone Marrow Stromal Stem Cells Transplantation in Congestive Heart Failure;
自体骨髓基质干细胞移植对充血性心衰心肌细胞影响的实验研究
9.
Effects of cAMP in Lymphocytes of Patients with Heart Failure on the Function of Peripheral T Help Lymphocyte
心力衰竭患者淋巴细胞cAMP对外周辅助T细胞功能的影响及意义
10.
Effect of testosterone on aging myocardial cell and the cell cycle regulation
睾酮对衰老心肌细胞及细胞周期调控因子的作用
11.
Lung as a Site of Synthesis and Storage of Cytokines in Experimental Congestive Heart Failure;
肺脏在心衰大鼠炎性细胞因子生成中的作用
12.
The Effect of Captopril on the Mice Cardiac Fibroblast Cell Senescence;
卡托普利对幼鼠心脏成纤维细胞衰老的影响
13.
Influence of Different Style Exercise on Aging Mice Cardiac Apoptosis
不同运动方式对衰老小鼠心肌细胞凋亡的影响
14.
Study of the changes of serum levels of cardiac function in patients with chronic heart failure
慢性心力衰竭中细胞因子的变化及其临床意义
15.
Changes of Caspase-3 Expression during Myocyte Apoptosis in the Rabbit Model of Congestive Heart Failure
Caspase-3在心衰兔模型细胞凋亡过程中的变化
16.
The role of p16~(INK4a) in the intervention of testosterone on senescence of murine cardiac myocytes
不同剂量睾酮干预心肌细胞衰老中p16~(INK4a)的表达
17.
Soluble Cell Adhesion Molecules and Congestive Heart Failure
可溶性细胞黏附分子与充血性心力衰竭
18.
Golgi complexes and centrosomes were especially rich in the cells treated with AMB and they did not show much decrepitude although the cells have aged.
特别是黄芪组细胞之高尔基复合体、中心体特别发达,虽细胞已衰老而它们却不甚衰老.