1) airway mucus secretion
气道黏液分泌
1.
Conclusion:The down-regulation of AQP5 expression may be involved in airway mucus secretion of asthma.
结论:AQP5蛋白表达下调可能参与了哮喘发病时气道黏液分泌的调节,IFN-γ可上调AQP5蛋白的表达,在哮喘气道黏液分泌中起治疗作用。
2) Airway mucus hypersecretion
气道黏液高分泌
1.
Effect of simvastatin on airway mucus hypersecretion in rats and its molecular mechanism;
辛伐他汀对大鼠气道黏液高分泌的影响和机制研究
2.
Airway mucus hypersecretion in rats is inhibited by macrolide antibiotics;
大环内酯类抗生素对大鼠气道黏液高分泌模型的干预作用研究
3.
Methods The rat airway mucus hypersecretion was induced by aerosolacrolein inhalation.
目的探讨丙烯醛诱导大鼠气道黏液高分泌发生的机制；了解PPARγ及其配体对气道黏液高分泌过程的调控作用；探讨PPARγ及其配体参与气道黏液高分泌调控的分子机制。
3) mucus overproduction
黏液分泌
1.
Objective: To evaluate the inhibitory effect of Yichuanping capsule (易喘平胶囊) on the airway mucus overproduction in asthmatic mice and its mechanism.
方法:将32只雄性BALB/c小鼠随机分为正常对照组、哮喘组、地塞米松组和易喘平胶囊组(n=8),制备卵蛋白致敏的哮喘模型,高碘酸希夫(PAS)法检测各组小鼠气道黏膜杯状细胞数量及黏液分泌情况,逆转录聚合酶链反应(RT PCR)检测肺组织黏液蛋白MUC5AC mRNA的水平,检测支气管肺泡灌洗液(BALF)中嗜酸粒细胞数及细胞总数,酶联免疫吸附法(ELISA)检测肺组织匀浆液中白细胞介素4(IL4)和γ干扰素(IFNγ)的含量。
5) airway mucus
气道黏液
1.
Inhibitory effect of specific inhibitor of p38 mitogen activated protein kinase on hyper secretion of airway mucus in asthma;
特异性p38丝裂原活化蛋白激酶抑制剂对哮喘气道黏液高分泌的抑制作用
补充资料：气道
气道
airway
气体出入的通道，多指气管和支气管。
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
参考词条