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1)  rHSG
大鼠增殖抑制基因
1.
Objective To explore relationship between expression of rats hyperplasia suppresion gene(rHSG) and delayed cerebral vasospasm(DCV) after experimental subarachnoid hemorrhage(SAH).
目的探讨大鼠蛛网膜下腔出血(SAH)后迟发性脑血管痉挛(DCV)和大鼠增殖抑制基因(rHSG)表达之间的关系。
2)  Hyperplasia suppressor gene
增殖抑制基因
1.
Objective To construct and identify the recombinant vector rHSG/pExpress-1 carrying rat hyperplasia suppressor gene (rHSG) which can express in eukaryote cells and which will provide the basis for further researching the mechanisms of adenoviral vector and working on tumorous genetherapy.
目的构建大鼠增殖抑制基因(rat hyperplasia suppressor gene,rHSG)的真核表达载体rHSG/pExpress-1并鉴定,为进一步构建腺病毒载体及恶性胶质瘤的基因治疗提供物质基础。
3)  Hyperplasia suppressor gene (HSG)
增殖抑制基因(HSG)
4)  proliferation inhibitor
增殖抑制因子
5)  growth inhibition
增殖抑制
1.
Growth inhibition effect of 5-azaC on the proliferation of HSG cells in vivo;
5-azaC对HSG细胞增殖抑制的实验研究
2.
3-[4,5-dimethylthiazol-2-yl]-2,5-diphenyltetrazolim bromide(MTT) assay was performed to study the growth inhibition rate of A-549 cells lines induced by As2O3 nanoparticles and flow cytometry(FCM) assay was used to examine their apoptosis rate induced by A.
应用MTT法研究As2O3纳米粒体外对细胞的增殖抑制作用;用流式细胞仪测定As2O3纳米粒及亚砷酸诱导细胞的凋亡率;用免疫组织化学半定量法检测As2O3纳米粒及亚砷酸处理细胞后Bcl-2、Bax、CD44v6和P53基因的表达改变。
3.
Purpose:To investigate the correlation between the anti-tumor activity,such as growth inhibition and apoptosis induction of a naphthoquinone analog,termed 2,3-dichloro-5,8-dihydroxy-1,4-naphthoquinone(DDN) and the expression pattern of p73.
相关性分析表明TAp73/DNp73表达比与细胞增殖抑制率之间呈正相关(r=0。
6)  Antiproliferation
增殖抑制
1.
Objective: To study the isomeric ratio change of lycopene after high pressure processing (HPP) and its antiproliferation activity.
采用噻唑蓝(MTT)法考察不同结构的番茄红素对雌激素受体阴性(ER-)人乳腺癌细胞MDA-MB-435S的增殖抑制作用。
补充资料:琥珀突变型抑制基因
分子式:
CAS号:

性质:又称琥珀突变型抑制基因。是一种能对琥珀密码子(终止密码子,UAG)作出反应,使氨基酸掺入位于mRNA琥珀密码子位点上的多肽链的抑制基因突变。所谓抑制基因突变是指完全或部分恢复初级突变所致功能丧失的次级突变,其突变位点不同于初级突变,不导致初级突变逆转,而是向双重突变体的野生型部分逆转。例如,大肠杆菌的一些菌株的琥珀校正基因,在琥珀密码子位点上可掺入酪氨酸、赖氨酸等一些其他氨基酸,此抑制基因突变发生在为tRNA编写的DNA顺序中的单个碱基取代,突变结果使tRNA的反密码子改变,能在核糖体上与mRNA的UAG配对,如单个碱基突变的酪氨酸tRNA,可读出链终止密码子UAG,如同UAG为酪氨酸的密码子那样。其完整多肽链的出现取决于特定的抑制基因特变,而功能性蛋白质的形成则取决于取代UAG的插入氨基酸。

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