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1)  congestive apoplexy
充血性卒中
2)  ischemic stroke
缺血性卒中
1.
Analysis of risk factors in patients with ischemic stroke;
缺血性卒中患者的危险因素分析
2.
Study on the pathogenesis hypothesis in ischemic stroke——study on the clinical basis for the pathogenesis hypothesis of "Qi deficiency blood stasis generating wind";
缺血性卒中病机假说——“气虚血瘀生风”临床依据研究
3.
Evidence-based Treatment for a Patient with Ischemic Stroke Accompanied by Hypertension and Atrial Fibrillation;
1例缺血性卒中合并高血压房颤患者的循证治疗
3)  hemorrhagic stroke
出血性卒中
1.
The levels of several endocrine hormones may change significantly after acute hemorrhagic stroke.
急性出血性卒中后机体多种内分泌激素水平会发生明显改变 ,其异常变化参与了出血性卒中的病理过程 ,对预后有明显影响。
4)  acute ischemic stroke
急性缺血性卒中
1.
The expression of Integrin CD11c/CD18 on the leukocytes of peripheral blood in patients with the acute ischemic stroke;
整合数CD11c/CD18在急性缺血性卒中患者外周血白细胞表达及意义
2.
Research progress of anticoagulation therapy of acute ischemic stroke;
急性缺血性卒中抗凝治疗研究进展
3.
Blood pressure management in acute ischemic stroke;
急性缺血性卒中血压处理策略
5)  acute hemorrhagic stroke
急性出血性卒中
1.
Objective To observe the effect of rhubarb in treating secondary damage of central nerve system (CNS) in rats with acute hemorrhagic stroke (AHS) and to explore the possible mechanism.
目的观察大黄对急性出血性卒中大鼠中枢神经二次损伤现象的治疗作用 ,并探讨其可能作用机理。
6)  Cerebral arterial thrombosis
缺血性脑卒中
1.
Clinical characteristics and relative risk factor of extracranial carotid arterial stenosis in patients with cerebral arterial thrombosis;
缺血性脑卒中患者颅外段颈动脉狭窄的临床特点及危险因素
2.
Clinical analysis of 12 cases of antihypertensive treatment complicated by cerebral arterial thrombosis;
老年原发性高血压降压治疗并发缺血性脑卒中12例临床分析
3.
Influence of different anticoagulant methods on GMP-140 and IL-6 on platelet surface and in plasma in patients with cerebral arterial thrombosis;
不同抗凝方法对缺血性脑卒中患者血小板表面及血浆内GMP-140和IL-6的影响
补充资料:脊髓缺血性疾病


脊髓缺血性疾病


  脊髓血管疾病的一种。脊髓缺血性血管病多由节段性动脉闭塞引起。如远端主动脉粥样硬化血栓形成引起肋间动脉或腰动脉闭塞。另外胸腹腔疾病、手术亦可损害脊髓造成梗塞,青年人常与血管畸形有关。由于供血不足,可以造成短暂性脊髓缺血,严重时可以发展成为永久性的脊髓损害。因其他疾病产生的短暂性血压过低,可以加重缺血,肢体远端无力与间歇性跛行为其特点。缺血早期,休息或使用扩血管药物可使无力现象缓解。病变继续发展则造成永久性损害,下肢无力不再为休息或药物所缓解,并可出现肌肉萎缩,共济失调和两点辨别觉消失,晚期出现排尿困难。治疗参见"动脉硬化"。
  
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