1) lobopodium,lobopodia
(复)叶足
2) lobopodia
(复数)叶足
4) etoposide
足叶乙甙
1.
PI3K/Akt/p27kip1 pathway mediates chemoresistance to Etoposide and Doxorubicin in gastric carcinoma cell line BGC-823 and its mechanism;
P13K/Akt/p27kip1通道介导胃癌细胞对阿霉素、足叶乙甙化疗的耐药性及其机制
2.
The Suppression Effect and bcl-2 Expression of K562 with Etoposide and Hyperthermia in Vitro;
热疗联合足叶乙甙对K562体外抑制效应及bcl-2的表达
3.
Effect of elemene and/or etoposide on the expression of survivin and apoptosis of retinoblastoma HXO-RB44 cells;
榄香烯乳联合足叶乙甙对视网膜母细胞瘤HXO-RB_(44)细胞凋亡及survivin表达的影响
5) VP16
足叶乙甙
1.
The Significance of TNFα Combined with VP16 on Growth of Lung Cancer Cell Line;
肿瘤坏死因子联合足叶乙甙对肺癌细胞株A549的毒性作用及临床意义
2.
Methods The inhibitory rate on cell proliferation of NSCLC in celecoxib group,DDP group,VP16 group,DDP with celecoxib group and VP16 with celecoxib group was detected by MTT assay.
方法应用噻唑蓝比色法(MTT)检测顺铂(DDP)、足叶乙甙(VP16)单独应用及与塞来昔布联合应用的增殖抑制率,并根据金氏公式计算Q值,评价两药的合用效应。
3.
Objective To discuss the role of survivin in the induction of apoptosis by VP16 in leukemia cells.
目的探讨生存素在化疗药物足叶乙甙(VP16)诱导白血病细胞凋亡中的作用。
6) etoposide
足叶乙苷
1.
AIM To study the role of survivin on the apoptosis of lung cancer cell line induced bycisplatin (DDP) and etoposide (VP16).
目的 探讨survivin基因在化疗药物顺铂 (DDP)和足叶乙苷 (VP16 )诱导的肺癌细胞凋亡中的作用。
2.
Objective:To explore the toxicity of As 2O 3 alone or in combination with daunorubicin (DNR), etoposide (VP16),or harringtanine (H) to sensitive leukamic cell line (K562) or to multidrug resistant (MDR) leukamic cell line (K562/A02).
As2 O3 分别与DNR、足叶乙苷 (VP16 )和三尖杉酯碱(H)联合应用时 ,对K5 6 2和K5 6 2 /A0 2的联合指数均大于 1。
3.
To study the possibility and the mechanism of K562 apoptosis induced by microwave and etoposide.
探讨微波和足叶乙苷体外诱导Ｋ５６２ 细胞凋亡的可能性及其机制。
补充资料：足叶乙甙
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