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1)  drug induced hepatitis
药物诱导性肝炎
2)  drug-induced autoimmune hepatitis
药物诱导的自身免疫性肝炎
3)  Con A-induced hepatitis
Con A-诱导性肝炎
4)  drug-induced hepatitis
药物性肝炎
1.
According to the history of taking ketoconazole, drug-induced hepatitis and subacute hepatic necros.
结合临床诊断为:酮康唑引起的药物性肝炎,亚急性肝坏死。
2.
Objective To know the etiological factors of prevention and cure of drug-induced hepatitis.
目的了解药物性肝炎的致病因素及防治。
3.
Objective: To observe the therapeutic effect on drug-induced hepatitis treated by combination of TCM and western medicine.
目的:观察中西药合用治疗药物性肝炎的疗效。
5)  Hepatitis induced by drugs
药物性肝肝炎
6)  severe drug-induced liver disease
重型药物性肝炎
1.
Objective To enhance the understanding to severe drug-induced liver disease by analyzing the etiology,clinical feature and related prognosis factors of 22 patients with severe drug-induced liver disease.
目的分析22例重型药物性肝炎的病因、临床特点及预后相关因素,以获得对重型药物性肝炎的认识。
2.
Objective:To enhance the understanding of severe drug-induced liver disease by analyzing the etiology,clinical feature and prognosis associated factor of 22 patients with severe drug-induced liver disease.
目的:分析22例重型药物性肝炎的病因、临床特点及预后相关因素,以加深对重型药物性肝炎的认识。
补充资料:自身诱导
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性质:一些药物重复使用后通过促进酶的合成速率或抑制酶的降解速率从而诱导或刺激了肝微粒体酶。酶诱导的结果加快了药物代谢,同时减低了药物的药理作用。其中有些药物如苯巴比妥和苯吡还可刺激酶对自身的代谢作用,使该药物代谢加速称为自身诱导。它成为药物耐药性的机制之一。

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