1) fatty inclusion
脂肪内含物
2) visceral adiposity
内脏脂肪含量
1.
Methods Eighty rats with different visceral adiposity were obtained through high fat feeding and weight loss method and were divided into normal model group,hypso-fat model group and weight relief group.
目的了解大鼠内脏脂肪含量对ghrelin分泌的影响。
3) IMF content
肌内脂肪含量
1.
Genetic polymorphism of H-FABP gene and correlation analysis with IMF content in porcine;
猪H-FABP基因遗传多态性及与肌内脂肪含量的相关分析
2.
By using 265 pigs from eight breeds including Duroc,Landrace,Large White,Neijiang,Rongchang,Hanjiang Black,Hanzhong White,Bamei and wild ones, the genetic variations of 5`-upstream region from and the second intron in porcine H-FABP gene were checked by PCR-RFLP molecular marker with HinfⅠ、HaeⅢ and MspⅠ,and effect of H-FABP gene on IMF content was then analyzed by least square analysis.
利用PCR RFLP(HinfⅠ、HaeⅢ和MspⅠ3种限制性内切酶)分子标记技术,检测了杜洛克猪、长白猪、大白猪、内江猪、荣昌猪、汉江黑猪、汉中白猪、八眉猪和野猪共计265头猪H FABP基因5′上游区和第二内含子区的遗传变异,并利用最小二乘模型分析了H FABP基因对猪肌内脂肪含量的遗传效应。
4) IMF
[英][,aɪ em 'ef] [美]['aɪ 'ɛm 'ɛf]
肌内脂肪含量
1.
Study on Single Nucleotide Polymorphism of H-FABP Gene and Its Association with IMF in Chicken;
鸡H-FABP基因多态性及其与肌内脂肪含量的相关研究
2.
The SNP was found only in Min pig with more intramuscular fat content(IMF) provide the support that H-FABP-proT224C could play an important role on IMF.
运用PCR-RFLP方法研究该位点在东北民猪、北京黑猪、长白猪、大白猪中的分布发现,只有肌内脂肪含量高的东北民猪中存在该突变,说明该突变位点很有可能与肌内脂肪含量相关。
3.
The developmental changes of FATP1 gene expression in SMBC usedβ-actin as internal standard control by the real-time quantitative RT-PCR method and the Correlation analysis between intramuscular fat(IMF), subcutaneous adipose thickness(SAT) a.
本论文以四川山地乌骨鸡、藏鸡和大恒优质肉鸡为研究对象,采用RT-PCR的方法扩增FATP1基因并对其cDNA序列和蛋白质结构进行了生物信息学的分析;运用实时荧光定量RT-PCR技术,以β-actin为内参基因,研究了四川山地乌骨鸡FATP1基因的发育性表达规律及其与肌内脂肪含量和皮脂厚的相关分析,比较了三个不同品种FATP1基因组织的表达差异。
5) intramuscular fat (IMF)content
肌内脂肪(IMF)含量
6) IMF of breast muscle
胸肌肌内脂肪含量
补充资料：脑病合并内脏脂肪变性综合征
脑病合并内脏脂肪变性综合征
En?cephalopathy with fatty degeneration of the viscera,Reye syndrome
本病又称“瑞氏综合征”。非炎症性急性脑肿胀，伴有以肝脏为主的内脏脂肪变性。主要发生在小儿时期，以4个月～5岁最多。病因和发病机制不明。诊断要点：①小儿病前一周内先有病毒感染，常见的为上感、水痘和胃肠炎；②较早出现反复呕吐及意识改变；③病情发展迅速，意识障碍或昏迷；④脑脊液正常；⑤肝功能检查有SGOT及SGPT明显增高、血氨增高，凝血酶原时间延长；⑥血清CPK增高；⑦无神经系统限局性体征；⑧神经系统表现和肝功能异常不能用其他疾病解释；⑨活检证实有肝脂肪变性；⑩肝大、过度换气、惊厥、低血糖、乳酸脱氢酶增高。治疗包括：积极支持疗法，抗惊厥、降颅压及肝功能衰竭的治疗等。
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
参考词条