1) experimental pneumonia
实验性肺炎
2) experimental silicosis
实验性矽肺
1.
Objective:According to silicosis model of mice reproduced by tratracheal injection method, our experiment studied antagonistic mechanism ofγ-Interferon (IFN-γ) against Tumor necrosis factor-α(TNF-α) and Transforming growth factor-β_1 (TGF-β_1) and the prophylactic therapeutic effect of,γ-interferon on experimental silicosis of mice, to prove up a new approach of treating lung fibrosis.
本实验通过气管内暴露法复制小鼠实验性矽肺纤维化模型,研究在动物体内γ-干扰素(γ-Interferon,IFN-γ)对致纤维化因子肿瘤坏死因子(Tumor necrosis factor-α,TNF-α)及转化生长因子-β_1(Transforming growth factor-β_1, TGF-β_1)的拮抗作用机制及其对实验性矽肺纤维化预防性治疗的效果,为治疗肺纤维化提供实验依据。
2.
ObjectiveAccording to silicosis model of rats reproduced by intratracheal injection method, our experiment studied antagonistic mechanism of - interferon (IFN- ) against Interleukin -4( IL -4) and Transforming growth factor - 1 (TGF- 1) and the prophylactic therapeutic effect of - interferon on experimental silicosis of rats, to prove up a new approach of treating lung fibrosis.
目的 本实验通过气管内非暴露法复制大鼠实验性矽肺纤维化模型，研究在动物体内γ-干扰素(γ-Interferon，IFN-γ)对致纤维化因子白介素-4(Interleukin-4，IL-4)及转化生长因子-β1(Transforming growth factorβ1，TGF-β1)的拮抗作用机制及其对实验性矽肺纤维化预防性治疗的效果，为治疗肺纤维化提供实验依据。
4) Experimental colitis
实验性肠炎
1.
[Methods] BALB/C mice were fed with 5% DSS for 7 days to induce experimental colitis, and macrophages in abdominal cavity and splenic cells were collected, the GSH, GSSG and GSH/GSSG in celiac macrophages and in splenic cells were determined by Glutathione Assay Kits.
结果DSS诱导的实验性肠炎小鼠腹腔巨噬细胞中的GSH较对照组降低,而GSSG较对照组增多,且GSH/GSSG比值明显减低。
5) experimental hepatitis
实验性肝炎
1.
Protective effects of Yangganqingrekeli on experimental hepatitis;
养肝清热颗粒对实验性肝炎的保护作用
6) Experimental gingivitis
实验性龈炎
补充资料：流行性喘憋性肺炎
流行性喘憋性肺炎
epidemic asthmatic pneumonia
又称“流行性毛细支气管炎”(epidemic bronchiolitis），是我国独特的流行性肺炎。本病的特点是：①在农村出现明显的爆发流行；②具有喘憋和发作性喘憋的特征；③具有毛细支气管炎及间质性肺炎的肺部表现；④主要侵犯婴幼儿。临床上大体可分为发病、喘憋、减轻、恢复几个阶段，不同程度的喘憋及发作性喘憋加重为本病的特征。病儿有咳嗽、鼻扇、呼吸急促等，发作性喘憋加重时明显烦躁不安、心率及呼吸更为加速，面色苍白(少数面红），口唇及指趾发绀，三凹征明显。肺部叩诊呈过清音，听诊有哮鸣音，少数尚有中、小水泡音，发作性喘憋极重时可有呼吸道梗阻，听不到呼吸音及喘鸣音。肝脏下缘因肺气肿而下降，重者且有肝肿大。喘憋多数在24～48小时内缓解。治疗参阅毛细支气管炎及支气管肺炎。
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
参考词条