1)  NF-kB
核因子-kB(NF-kB)
2)  nuclear factor-kB(NF-kB)
核因子kB(NF-kB)
3)  Nuclear factor-κB
核因子-κB
1.
Effects of nuclear factor-κB on corneal transplant rejection;
核因子-κB参与角膜移植排斥反应的实验研究
2.
Effect of nuclear factor-κB activation on expression of proinflammatory cytokines in rat lung tissues in early stage of burn injury;
核因子-κB活化对大鼠烧伤早期肺组织表达促炎细胞因子的作用
3.
Relationship between nuclear factor-κB and apoptosis of hepatocyte in rats with septic shock;
核因子-κB与感染性休克大鼠肝细胞凋亡的关系
4)  NF-κB
核因子kappaB
1.
The Changes of Nuclear Factor Kappa B(NF-κB) after Cerebral Ischemia Reperfusion in Rats;
脑缺血再灌注后实验大鼠核因子kappaB的变化
5)  NF-κB
核因子-κB
1.
Expression of NF-κB and its effect on IL-4 in cultured cells from bleomycin-injured lung tissue in mice;
核因子-κB在博莱霉素损伤小鼠肺组织培养细胞中的表达及对白细胞介素4的影响
2.
The effect and significance of lightusrazine on the expression of NF-κB in primary cultured neural cells after ischemia-reperfusion injury;
川芎嗪对原代神经细胞缺血再灌注后核因子-κB表达的影响及其意义
3.
Effect of p38 MAPK inhibitor pretreatment on NF-κB in pulmonary in rats with acute lung injury;
p38MAPK抑制剂对急性肺损伤大鼠肺组织核因子-κB表达的影响
6)  Nuclear factor-κB
核因子κB
1.
Objective To investigate the relations between nuclear factor-κB(NF-κB)and the development of lipopolysaccharide(LPS)-induced acute kidney injury(AKI),and to explore the effect of ulinastatin(UTI)on AKI.
目的研究核因子κB(nuclear factor-κB,NF-κB)与内毒素(lipopolysaccharide,LPS)诱导的大鼠急性肾损伤(acute kid-neyinjury,AKI)发病的关系及初步探讨乌司他丁(ulinastatin,UTI)对大鼠AKI时肾脏的保护作用。
2.
Objective To explore the mechanism of traditional Chinese medicine(TCM) treatment according to syndrome differentiation in treating chronic obstructive pulmonary disease(COPD) by observing the changes of nuclear factor-κB(NF-κB) and γ-glutamylcysteine synthetase(γ-GCS) expression levels in rats.
目的通过观察慢性阻塞性肺疾病(COPD)大鼠核因子κB和γ-谷氨酰半胱氨酸合酶(γ-GCS)水平的变化,探讨中医药治疗COPD的机理。
3.
Objectives The aim of the current study was to investigate the nuclear factor-κB(NF-κB) binding activity and expression of metalloproteinase-9(MMP-9) in the injured brain and to explore the potential role of NF-κB and MMP-9 in the secondary brain injury after traumatic brain injury(TBI).
目的研究创伤性脑损伤(TB I)后损伤区脑组织核因子κB(NF-κB)和基质金属蛋白酶-9(MMP-9)的表达,探讨其在继发性脑损害中的作用机制。
参考词条
补充资料:红色碱基KB
分子式:C7H9Cl2N
分子量:178.05
CAS号:6259-42-3

性质:该品为灰白色粉状物,贮藏后颜色变深。纯品(游离胺)熔点为20-22℃,沸点238.5℃。极易溶于乙醇、乙醚等有机溶剂;溶于热水并随水蒸气蒸发。其盐酸盐为无色结晶,于80℃升华,在247-249℃分解。与色酚AS及AS-D偶合成鲜艳红色。

制备方法:采用对甲苯胺为原料、经硝化、重氮化、氯化、还原、成盐,再经过滤、干燥而得。产品中色基含量≥95%,水分含量≤0.5%,不溶于盐酸的杂质含量≤0.2%。原料消耗(kg/t)对甲苯胺 780硫酸 4700硝酸 680硫酸铜(64%) 614硫化钠(60%) 3200盐酸(31%) 1000精盐 3000

用途:主要用于棉、丝和锦纶纤维织物的染色和印花的显色剂,并可用于制备快色素。与色酚AS及色酚AS-D偶合呈鲜艳红色。

说明:补充资料仅用于学习参考,请勿用于其它任何用途。