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1)  hyperthyroidism hepatic dysfunction
甲亢性肝损伤
1.
Objective: To explore the relation between hyperthyroidism and hepatic dysfunction and to study the prophylaxis and treatment of hyperthyroidism hepatic dysfunction with Jia Kang Ning.
目的:探讨甲状腺功能亢进患者对肝功能的影响以及中药甲亢宁对甲亢性肝损伤的防治作用。
2)  Hepatic Injury
甲亢肝损害
1.
The Association between Graves Disease of Hepatic Injury and Polymorphism in Transporter Associated with Antigen Processing 1 Gene;
Graves甲亢肝损害与抗原肽运载体1基因多态性关联性的研究
3)  Hyperthyroid hepatopathy
甲亢性肝病
1.
Objective:To investigate clinical feature of patients with hyperthyroid hepatopathy.
目的:探讨甲亢性肝病患者肝损害的临床特点。
4)  liver injury
急性肝损伤
1.
Protective effect of celery root extract on acute liver injury induced by CCl_4 in rats;
芹菜根提取物对四氯化碳所致大鼠急性肝损伤的保护作用
2.
Study on protective effects of astaxanthin on acute chemical liver injury in mice
虾青素对四氯化碳致小鼠急性肝损伤保护作用
3.
Objective To explore the anti-inflammation and analgesia effect of Radix Fici Hirtae aqueous extract, and its protection effect on acute liver injury.
目的探讨五指毛桃水提液的抗炎镇痛及对急性肝损伤的保护作用,为临床运用提供依据。
5)  Acute hepatic injury
急性肝损伤
1.
An experimental Study of Qin Long Granule on Acute Hepatic Injury in Rats;
芹龙颗粒抗急性肝损伤的实验研究
2.
Effects of alprostadil for injection in inhibition of IL-18 level for rats with D-galactosamin-induced acute hepatic injury;
前列地尔对D-氨基半乳糖致急性肝损伤大鼠肝匀浆IL-18水平升高的抑制作用
3.
Protective effect of Xiaoyan Lidan Tablet on acute hepatic injury in rats;
消炎利胆片防治大鼠急性肝损伤的实验研究
6)  Chronic hepatic injury
慢性肝损伤
1.
Effects of Qichai Fugan Tang(芪柴复肝汤) on chronic hepatic injury by CCl_4 in rat;
芪柴复肝汤对CCl_4所致大鼠慢性肝损伤的影响
2.
The roles of the Chinese traditional medicine Heluoshugan capsule in rats chronic hepatic injury and the mechanism were investigated.
探讨中药制剂和络舒肝胶囊的抗慢性肝损伤作用及作用机制。
3.
Objective:To validate the protective role of Jidesheng Sheyao in antagonizing chronic hepatic injury caused by carbon tetrachloride (CCl4).
目的:观察季德胜蛇药对四氯化碳(CCl4)所致小鼠慢性肝损伤的保护作用。
补充资料:肝外阻塞性胆汁性肝硬化


肝外阻塞性胆汁性肝硬化


又称"继发性胆汁性肝硬化。*胆汁性肝硬化的一个类型,继发于肝外胆管阻塞而引起的肝硬化。主要由总胆管结石、手术后胆管狭管、胰头癌、先天性胆管闭锁或缺如等原因所造成。在梗阻上端的胆道由下而上地逐渐扩大迂曲,胆汁淤积,肝内小胆管扩大而破裂,使肝血管受压,胆汁外渗,肝细胞发生缺血、坏死,纤维组织增生,最后形成肝硬化。临床表现为黄疸、肝脾肿大,皮肤粘膜出血倾向,血胆固醇下降等,晚期表现同*肝硬化。解除肝外阻塞原因,本病有治愈的可能性。
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