1)  U251 cells
U251 细胞
1.
To investigate the ability of recombinant human interferon-gamma(IFN-γ) to upregulate major histocompatibility complex class I(MHC class I)molecules expression on the membrane of human glioblastoma multiforme(GBM)U251 cells in vitro.
观察重组人IFN-γ对体外人多形性胶质母细胞瘤(GBM)U251 细胞胞膜MHC-I 类分子表达的影响;2。
2)  U251 cell line
U251细胞
1.
Knockdown of STAT3 expression by siRNA induces apoptosis in U251 cell line;
STAT3 siRNA引起U251细胞凋亡的研究
2.
To construct erythroblastic leukemia viral oncogene homolog 2(ErbB2)small interference RNA (siRNA)expression vector and to study its effect on U251 cell line proliferation and apoptosis combining with ~(60)Coγ-iradiation.
构建特异性抑制白血病病毒致癌基因同源体2(ErbB2)的小干扰RNA(siRNA)表达载体,并检测联合~(60)Coγ照射对U251细胞增殖抑制及促进凋亡作用。
3)  U251 cell
U251细胞
1.
Construction of phage display antibody library of the special antigen responding to serum starved U251 cell;
U251细胞血清饥饿特异抗原的ScFv噬菌体抗体库构建
2.
Inhibition effect of recombinant mutant human TNF-α combined with cisplatin on human glioma U251 cells;
重组改构人TNF-α联合顺铂对人胶质瘤U251细胞的生长抑制作用
3.
An Initial Study on the Effect of p53 Gene on the Expression of Its Downstream Genes in U251 Cell Line;
p53基因对U251细胞中基因表达影响的初步研究
4)  U251 cells
U251细胞
1.
Blockage of U251 cells in G_0/G_1 through MAPK signaling pathway by LRRC4;
LRRC4通过MAPK信号通路阻滞U251细胞于G_0/G_1期
2.
Inhibition effect of Bcl-2 small hairpin RNA on the growth of U251 cells via transferrinreceptor-mediated delivery;
转铁蛋白受体介导的Bcl-2 shRNA对U251细胞生长的抑制
3.
CS-GAG of proteglycan NG2 modulate the migration of NG2 transfected U251 cells;
蛋白聚糖NG2中硫酸软骨素糖胺聚糖链调节稳定转染NG2的U251细胞迁移能力
5)  U251 cells
U251细胞株
6)  U251 cell line
U251细胞系
1.
OBJECTIVE The aim of the first part was to sort subpopulation of CD133+ and CD133- cells in the human Glioblastoma Multiforme(GBM)U251 cell line.
目的 第一部分实验旨在运用磁珠分选技术(magnetic cell sorting,MACS)分选人多形性胶质母细胞瘤U251细胞系中的CD133+和CD133-亚群细胞;比较两个亚群细胞在增殖、分化及体内致瘤性等方面的差异,重点探讨CD133+细胞生物学特性;评价CD133作为脑肿瘤干细胞表面标志物的特异性与磁珠分选系统分选脑肿瘤干细胞的效率及特异性。
参考词条
补充资料：Fluorescent Brightener 251
分子式：C36H32N12Na4O14S4
分子量：1076.93
CAS号：16324-27-9

性质：

说明：补充资料仅用于学习参考，请勿用于其它任何用途。