1)  rAd/p53
rAd/p53
1.
Methods: The cell growth inhibition and apoptosis of rAd/p53 and TNF were detected by MTT and flow cytometry.
目的:探讨重组人p53腺病毒注射液(rAd/p53)联合肿瘤坏死因子(TNF)对荷瘤小鼠体内外的乳腺癌细胞Ca761的抑制效果。
2.
Objective To explore the effect of rAd/p53 to breast cancer of the mice by different administrations.
目的探讨重组人p53腺病毒注射液(rAd/p53)不同用药方式对荷瘤小鼠乳腺癌的治疗效果。
2)  rAd-p53
重组人p53腺病毒注射液
3)  rAd-p53
重组人p53腺病毒
1.
Clinical Observation of Advanced Pulmonary Carcinoma Injected with rAd-p53;
瘤内注射重组人p53腺病毒治疗晚期肺癌临床试验观察
2.
Study of Treatment of Malignant Pleural Effusion in Patients with rAd-p53;
重组人p53腺病毒治疗恶性胸腔积液的研究
3.
Objective To understand the inhibition and IC50 (50% inhibiting concentration) of the recombinant adenoviral p53 gene (rAd-p53) for the colorectal cancer cells in vitro and to guide clinical practice.
目的为了了解重组人p53腺病毒注射液(商品名“今又生”)对大肠癌-174细胞株的体外抑制作用及其半数致死量(IC50),更好地指导临床应用。
4)  rAd-p53
rAd-p53
1.
Objective:To investigate the best time sequence combined treatment with rAd-p53 (Gendicine) and cisplatin(DDP) to the lung adenocarcinoma cells in vitro.
目的:研究rAd-p53(商品名:今又生)与顺铂以不同序贯方式联合应用对肺腺癌细胞的增殖抑制作用,明确rAd-p53与顺铂联合的协同效应关系和最佳作用时序。
2.
In this paper investigating inhibitory effect and radiation sensitization of recombinant adenovirus en-coding human p53 tumor suppressor gene (rAd-p53) on human lymphoma cell lines has been performed by MTT,Laser confocal scanning microscope (LCSM) and flow cytometry.
本研究利用四氮唑盐(Methyl thiazolyl tetrazolium,MTT)比色法、激光共聚焦型显微镜扫描技术和流式细胞术分析重组人p53腺病毒注射液(商品名今又生,rAd-p53注射液)对人淋巴瘤细胞的抑制作用及其辐射增敏性。
5)  rAd-p53
重组腺病毒携带P53
6)  Adp53
Adp53
1.
METHODS Human hepatocarcinoma Hep3B cells were transfected with a replication-defective advenovirus encoding(Adp53),and Wnt signaling pathway function changes with key factor β-catenin.
方法:将携带p53基因的复制缺陷型腺病毒载体(Adp53)导入到p53完全缺失的人肝癌细胞株Hep3B中,并以Wnt通路的关键因子β-链接蛋白(-βcatenin)的改变为功能指标评价Wnt通路的变化。
2.
Methods Human hepatocarcinoma Hep3B cells were transfected with a replication-defective advenovirus encoding(Adp53),HepG2 cell lines(wild p53) were transfected with p53 antisense ODN and Wnt signaling pathway function changes with key factor β-catenin.
方法将携带p53基因的复制缺陷型腺病毒载体(Adp53)导入到p53完全缺失的人肝癌细胞株Hep3B中,并以p53反以寡核苷酸阻断p53阳性细胞p53的表达,以W nt通路的关键因子-βcaten in的改变为功能指标评价W nt通路的变化。
3.
To examine whether p53 has any regulatory effects on the expression of Dickkopf-1 ( Dkk-1), a Wnt pathway inhibitor, Hep3B hepatoma cells, were transfected with areplication-defective adenovirus encoding p53(Adp53), and then dickkopf-1 (DKK-1) mRNA expression was detected by RT-PCR.
将携带p53基因的复制缺陷型腺病毒栽体(Adp53)导入到p53缺失的人肝癌细胞株Hep3B中,以RT-PCR技术检测p53对DKK-1表达的调节作用。
参考词条
补充资料：radium D RaD
分子式：
CAS号：

性质：历史名称，是核素210Pb。放射性核素，氡子体之一，属铀系成员，主要发生β-衰变(约100％)，半衰期为22.3a，生成210Bi(RaE)；也能发生α衰变(～2×10-6％)，生成206Hg。在氡子体中，其半衰期最长，它的积累和衰减都需较长时间，其多少一般反应长期氡浓度变化的稳定值。与1t 238U相平衡的质量为4.36×10-3g。

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