1) Pulmonary damage/ pulmonary toxicity
肺损伤/肺毒性
2) acute lung injury
急性肺损伤
1.
Accommodation of Qidonghuoxue decoction on disbalance of oxidation/anti-oxidation system in rats with acute lung injury induced by Oleic Acid;
芪冬活血饮对油酸致急性肺损伤大鼠氧化/抗氧化失衡的调节作用
2.
New development of blood transfusion-related acute lung injury(TRALI);
输血相关性急性肺损伤及其新进展
3.
Effects of inhalation of endothelin-A receptor antagonist on experimental acute lung injury;
吸入内皮素A受体阻滞剂对实验性急性肺损伤的影响
3) acute lung injury (ALI)
急性肺损伤
1.
Objective To investigate the role of 3-quinuclidinyl benzilate (QNB) on acute lung injury (ALI) induced by PFIB inhalation,and to unveil the mechanism of hemorheological changes.
目的评价二苯羟乙酸-3-喹咛环酯(3-quinuclidinylbenzilate,QNB)对全氟异丁烯(Perfluoroisobutylene,PFIB)吸入急性肺损伤的防治效果,并初步探讨其血液流变学作用机制。
2.
Recent studies have demonstrated that in acute lung injury (ALI) or acute respiratory distress syndrome (ARDS), the extrinsic coagulation systems are activated by pathogen and its’metabolic product, especially by leukocytes emerged in the early stage of inflammatory reaction.
近年来的研究证明,在急性肺损伤/急性呼吸窘迫综合征(ALI/ARDS)的发病初期,病原体及代谢产物(如内、外毒素),尤其是炎症反应早期出现的细胞因子,可激活外源性凝血途径,导致弥漫性血管内凝血(DIC)。
3.
Background: Acute lung injury (ALI) and Acute respiratory distress syndrome(ARDS) refer to acute and progressing respiratory failure(RF) that is induced by various kinds of etiological factor except cardiogenic factors.
背景:急性肺损伤(Acute lung injury ,ALI)和急性呼吸窘迫综合征(Acute respiratory distress syndrome,ARDS)是指由心源性以外的各种肺内外致病因素所导致的急性、进行性缺氧性呼吸衰竭。
4) ALI
急性肺损伤
1.
The infuluence of Shenmai effects on Chrome level to the rat when ALI;
铬在大鼠急性肺损伤中变化及参脉的干预作用
2.
The influence of Chinese herbal to oxy-radicals of ALI;
中药在急性肺损伤中抗氧自由基的作用
3.
The influence of Vitamin C on CRP of ALI of big rats;
维生素C对大鼠急性肺损伤CRP水平的影响
5) Acute lung injury(ALI)
急性肺损伤
1.
The clinical result of the lung protective ventilation and Ulinastatin intravenous for Acute lung injury(ALI);
急性肺损伤行肺保护性通气加乌司他丁静滴的临床效果
2.
Objective:To observe the fibroproliferation occurs early in the acute lung injury(ALI)caused by lipopolysacharide(LPS).
目的:观察脂多糖(LPS)致大鼠急性肺损伤(ALI)早期是否存在肺纤维增生,探讨其在ALI发病机制中的作用。
3.
Objective:To investigate the suppressive effects of bilirubin on neutrophilic infiltration as well as the mechanisms of bilirubin in the prvention in acute lung injury(ALI).
目的:探讨胆红素对急性肺损伤(ALI)的保护作用及其对中性粒细胞肺浸润的抑制作用。
补充资料：肺毒性
肺毒性
由化疗药引起的肺毒性表现为弥漫性间质性肺炎，最终导致肺纤维化。最常引起肺毒性的化疗药物是博莱霉素。平阳霉素、甲氨蝶呤、马利兰、环磷酰胺、瘤可宁、丝裂霉素等长期应用也可引起肺纤维化。肺毒性的原因尚不清楚，可能是①肺从循环中清除内源性有毒物质，如化疗药，使肺内有毒药物浓度相对增加；②肺能进行药物的代谢转化；③有些药物对肺泡壁敏感，使之产生类六甲铵样作用而诱发肺纤维化；④肺毒性也是一种过敏反应。防治：①严格掌握药物适应证；②对用博莱霉素的病人每3～6个月做一次肺功能检查；③博莱霉素用药量应限制在360mg以下；④发现肺毒性及时停药。
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
参考词条