3) cardiomyocyte hypertrophy
心肌细胞肥大
1.
Effects of exogenous Apelin on cardiomyocyte hypertrophy;
Apelin对心肌细胞肥大的影响
2.
Study of cross-talk between stat family and smads pathway mediated cardiomyocyte hypertrophy in cultured neonatal rat in vitro
新生大鼠心肌细胞通过STAT和Smads信号交叉对话介导心肌细胞肥大的体外研究
3.
OBJECTIVE To observe effects of Sodium tanshinone IIA sulfonate(STS) on angiotensin II(Ang Ⅱ)-induced cardiomyocyte hypertrophy and expressions of phosphorylated p38(p-p38) and mitogen-activated protein kinase phosphatase-1(MKP-1).
目的观察丹参酮ⅡA磺酸钠(Sodium tanshinoneⅡA sulfonate,STS)对血管紧张素Ⅱ(angiotensinⅡ,AngⅡ)诱导的心肌细胞肥大反应及p-p38,MKP-1表达的影响。
4) Cardiac hypertrophy
心肌细胞肥大
1.
The Effect of Erythropoietin on cardiac hypertrophy
促红细胞生成素促心肌细胞肥大的实验研究
2.
Objective: to study the inhibition effect of simvastatin on cardiac hypertrophy induced by myocardial infarction,and to investigate whether simvastatin prevent cardiac hypertrophy through attenuation of RhoA/ROCK and cyclin D1.
目的:观察辛伐他汀对大鼠心肌梗死后心肌细胞肥大的抑制作用,探讨小G蛋白RhoA激酶(ROCK)/细胞周期蛋白D1(Cyclin D1)途径在辛伐他汀抑制心肌细胞肥大中的信号转导作用。
3.
However,little is known about the role of Epo in the development of cardiac hypertrophy.
有关Epo对心肌细胞肥大影响的报导在国内外非常有限。
5) human adult hypertrophic cardiomyocytes
人肥大心肌细胞
1.
The establishment of the model for hypoxia-reoxygenation induced apoptosis of culturing human adult hypertrophic cardiomyocytes;
缺氧/复氧诱导培养人肥大心肌细胞凋亡模型的建立
6) hypertrophic cardiomyocytes
肥大心肌细胞
1.
Objective To investigate the hypertrophic cardiomyocytes apoptosis induced by hypoxia and hypoxia /reoxygenation.
结果肥大心肌细胞在缺氧8h时即出现显著的细胞凋亡。
2.
In the study, we investigated the change rule of the cytoskeleton and biomechanics of hypertrophic cardiomyocytes and furthermore illuminated.
本研究目的就在于观察肥大心肌细胞骨架、生物力学特性的变化规律,并进一步阐明细胞骨架改变对肥大心肌细胞生物力学特性的具体影响及其调节机制。
补充资料：肥厚型心肌病
肥厚型心肌病
hypertrophic cardiomyopathy
原发性心肌病的一型。多见于10岁左右男孩。有人认为本病为常染色体显性遗传。心脏病变主要为室间隔及左室壁心肌肥厚，室间隔更为显著。可分为梗阻型及非梗阻型。早期症状为运动后呼吸困难，心慌、心绞痛、头晕或晕厥。心力衰竭罕见，但可猝死。心脏检查可见心界向左扩大，有抬举性心尖搏动，胸骨左缘下端及近心尖部听到收缩期喷射状杂音等。治疗方面应限制病儿剧烈运动。洋地黄、异丙基肾上腺素等使心脏收缩力加强的药物，可加重流出道梗死，应避免使用。β-肾上腺素能阻滞剂心得安治疗本病，可缓解症状。内科治疗无效者，可外科手术切除肥厚的心肌。
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
参考词条