1) neurongl discharge variability
神经元放电变异性
2) Neuron Discharge
神经元放电
1.
A Wavelet-Based Method of Neuron Discharge Extraction for Parkinson s Patient;
为此提出一种新的非线性方法提取神经元放电,利用多分辨小波变换对信号分解,对有用信息分量系数平方,再对信号重构。
3) denatured neurons
变性神经元
1.
Objective To establish a experimental model for evaluating the remedy for denatured neurons after forebrain ischemia reperfusion.
目的以海马CA1区细胞形态为观察对象,筛选评价脑缺血再灌注损伤后对变性神经元救治干预的实验平台。
2.
Objective: To establish a experimental model for evaluating the effect of remedy for denatured neurons after cerebral ischemia-reperfusion.
目的:探索以变性神经元为观察对象,适用于脑缺血再灌注后干预实验的动物模型。
4) Neuronal degeneration
神经元变性
1.
In order to test Fluoro-Jade C staining method for detecting neuronal degeneration in the midbrain substantia nigra of animal models induced by neurotoxicant 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP) or kainic acid (KA).
为探讨Fluoro-JadeC荧光染色检测神经毒物MPTP和红藻氨酸(KA)损伤致中脑黑质神经元变性死亡的方法,本研究采用小鼠腹腔注射MPTP或黑质定位注射KA制备黑质损伤模型,然后进行Fluoro-JadeC染色标记和计数分析黑质内变性死亡神经元。
2.
The mechanism of neuronal degeneration or apoptosis includes two major cell death signaling pathways which are mitochondrial death pathway and death receptor pathway.
研究背景阿尔茨海默病(Alzheimer\'s disease,AD)的典型神经病理特征是患者脑内出现以β-淀粉样蛋白(amyloid beta peptide,Aβ)为主要成分的神经元外老年斑(senile plaque,SP)、神经元内神经原纤维缠结(neurofibrillary tangles,NFTs)、广泛的神经元变性消失。
5) Neurodegeneration
神经元变性
1.
Electron microscopic observation on trauma-induced neurodegeneration in newborn rat brain;
新生鼠脑外伤后神经元变性的电镜观察
2.
In order to elucidate the neuropathogenesis and seek for intervention methods for Niemann-Pick disease type C (NPC), histological, immunohistochemical and immunoblotting methods were performed to detect the neuropathological and cytoskeletal changes as well as the presence of mitotic markers which are considered to be the early markers of neurodegeneration in a murine model of NPC.
针对这一发现可设计干预方案来阻止神经元变性过程。
3.
To investigate pathogenesis of pathological axonal spheroids in Niemann-Pick disease type C(NPC), a murine model of NPC, the Balb/c nih npc-1 mouse strain, has been studied using immunohistochemicaland immunoblot techniques, to explore activation of cdk4/cyclin D1/Rb2 pathway and its association withneurodegeneration and hyperphosphorylated neurofilament.
以Balb/cnihnpc-1小鼠模型为对象,使用免疫组织化学和免疫印迹技术来研究cdk4/cyclinD1/Rb2通路的激活与神经元神经丝蛋白的过度磷酸化及神经元变性的相互关系,探讨C型Niemann-Pick病(NPC)病理性球状神经轴突形成的机制。
补充资料：神经元形态中枢神经系统内轴突髓鞘的形成示意图
李瑞端绘
[图]
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
参考词条