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1)  galactosidaseT]
β-半乳糖甘酶
2)  endo-β-mannanase
内源β-半乳甘露聚糖酶
1.
Activity and distribution of endo-β-mannanase during germination of watermelon seeds and its relationship with germination rate;
西瓜种子内源β-半乳甘露聚糖酶活性与分布以及与萌发速度的关系
3)  β-galactosidase
β-半乳糖苷酶
1.
Selection of Strains Highly Producingβ-galactosidase with Complex Mutation;
采用复合诱变方法选育高产β-半乳糖苷酶菌株
2.
Fermentation Conditions for Production of Transglycosylating β-galactosidase by Enterobacter cloacae B5;
Enterobacter cloacae B5产转糖基β-半乳糖苷酶发酵条件优化
3.
Study on Activity of β-galactosidase Produced by Lactobacillus acidophilus with Ultrasionic Cell-break Method;
超声波细胞破碎法检测嗜酸乳杆菌β-半乳糖苷酶活力的研究
4)  β-D-galactosidase
β-D-半乳糖苷酶
1.
Correlation between β-D-galactosidase and gelatinase B activity and progress of colorectal carcinoma;
β-D-半乳糖苷酶和明胶酶B活性与大肠癌进展的相关性
2.
Study on the immobilization of β-D-galactosidase on chitosan gel;
聚糖凝胶固定β-D-半乳糖苷酶方法研究
3.
Synthesis of Galacto-oligosaccharide from Lactose Catalyzed by K.fragilis LFS-8611 β-D-galactosidase;
脆壁克鲁维酵母(Kluyveromyces Fragilis)LFS-8611 β-D-半乳糖苷酶催化合成低聚半乳糖
5)  beta-galactosidase
β-半乳糖苷酶类
6)  β-galactosidase
β-半乳糖酸苷酶
1.
This paper mainly studies the effect of ultrafine brucite powders on the growth progress of E coli by analysing major chemical composition of ultrafine brucite mineral powders and determining the changes of number of colony,pH,glucose(GLU),β-galactosidase,Mg~(2+) after the powders acted with E coli ATCC25922 at different time.
通过分析超细纤维水镁石的主要化学成分,测定其与大肠杆菌ATCC 25922相互作用不同时间后,菌落数、培养基pH值、葡萄糖(GLU)、β-半乳糖酸苷酶以及M g2+等的变化,来研究其相互作用影响。
2.
This paper mainly studies the effect of ultrafine brucite powders on the growth progress of E coli by analysing major chemical composition of ultrafine brucite mineral powders and determining the changes of number of colony,pH,glucose (GLU), β-galactosidase, Mg~(2+) after the powders acted with E coli ATCC25922 at different time.
通过分析超细纤维水镁石的主要化学成分,测定其与大肠杆菌ATCC25922相互作用不同时间后,菌落数、培养基pH值、葡萄糖(GLU)、β-半乳糖酸苷酶以及Mg~(2+)等的变化,来研究其相互作用影响。
补充资料:1-磷酸半乳糖尿苷转移酶缺乏


1-磷酸半乳糖尿苷转移酶缺乏


  通常所称半乳糖血症即指此类,最为常见。本病呈常染色体隐性遗传。肝、肾、晶体及脑组织是主要受累器官。由于此酶缺乏,前质1-磷酸半乳糖及半乳糖堆积。细胞内过多的1-磷酸半乳糖被认为是毒性物质,是引起临床症状的主要原因。婴儿出生时正常,喂奶后出现呕吐、拒食、不安、腹泻,继而肌张力低下、黄疸及肝肿大。母乳乳糖含量较高症状较重。若病婴继续摄入乳糖,可出现肝硬化、低血糖等。病儿常伴营养不良、白内障等。体格及智能障碍明显,治疗要尽早摒除半乳糖,婴儿期可用豆浆代替。经治疗白内障、肝硬化可逆转。
  
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