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1)  transmembrane helical region
跨膜螺旋区
2)  transmembrane helical segments
跨膜螺旋区段
1.
Increasing protein sequences from the genome project require theoretical methods to predict transmembrane helical segments(TMHs).
基因组计划所产生的大量蛋白质序列迫切需要从理论上预测跨膜螺旋区段。
3)  transmembrane helix
跨膜螺旋
1.
9% similarity; the secondary structure of protein has 9 α helix and 11 β sheet for hNDR2, 9 α helix and 12 β sheet for mNDR2; the hNDR2 and mNDR2 protein belong to α/β folding class; both of them have one transmembrane helix structure.
9%的相似性 ,hNDR2蛋白质二级结构有 9个α螺旋和 11个 β叠片 ,属于α/β折叠类蛋白 ;mNDR2蛋白质二级结构有 9个α螺旋和 12个 β叠片 ,属于α/β折叠类蛋白 ;两者都有一个跨膜螺旋结
4)  transmembrane helices structure
跨膜螺旋结构
1.
The transmembrane helices structure of domestic goose Cyt b gene and sequence variation in every zone first were analysed and reported by way of studying mitochondrial genome in the paper.
通过对家鹅线粒体基因组的研究,首次报道了家鹅线粒体Cytb的跨膜螺旋结构,并对家鹅Cytb跨膜螺旋结构及各区域序列的变异特征进行了分析。
5)  transmembrane domain
跨膜区
1.
The chromosomal location of AD7C-NTP gene and the prediction of transmembrane domains of its deduced protein;
AD7C-NTP基因的染色体定位及其产物的跨膜区预测
2.
Objective To investigate the mutation at the transmembrane domain of fibroblast growth factor receptor 3 (FGFR3) nucleotide 1138 site for identifying the major pathologic mechanism of achondroplasia (ACH) and to evaluate the efficacy of denaturing gradient gel electrophoresis(DGGE) method for screening the point mutations.
目的 验证成纤维细胞生长因子受体 3(fibroblastgrowth factor receptor3,FGFR3)跨膜区1138位核苷酸为先天性软骨发育不全突变热点及用变性梯度电泳方法筛查的效果。
3.
Methods: Eight recombinants were constructed in order to study whether the expression of rhZP3 will be influenced by the following factors,which include retaining of the transmembrane domain of rhZP3,mutation of the potential sites of protease,and the position of His-tag.
方法:构建8个重组rhZP3的差异表达质粒在毕赤酵母中进行诱导表达,研究跨膜区保留与否、潜在酶切位点突变与否及多聚组氨酸纯化标签设计的位置是否影响rhZP3的表达情况。
6)  coiled hollow fiber membrane
螺旋形膜
补充资料:跨膜电位
分子式:
CAS号:

性质:膜(主要指生物膜)两侧的电位差。它取决于膜内所带的电荷与膜两侧所带的电荷,后一种包含膜表面和相邻溶液所带的电荷。例如在安静状态的神经膜的跨膜电位大约为-75mV,表示细胞内侧相对于外侧低75mV,膜内侧显负电性而外侧显正电性。当神经细胞受激时,跨膜电位从-75mV变成了+50mV,膜内侧显正电性而外侧显负电性,此时将使膜内侧过多的阳离子挤出到外侧。

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