1) human bronchial epithelial cell line BEP2D
人支气管上皮细胞BEP2D
3) human bronchial epithelial cells
人支气管上皮细胞
1.
Effect of calcitonin-gene-related peptide on MMP-9 production in human bronchial epithelial cells;
降钙素基因相关肽对人支气管上皮细胞分泌MMP-9的影响
2.
Malignant transformation of immortalized human bronchial epithelial cells with thrombin;
凝血酶诱发人支气管上皮细胞恶性转化
3.
Influence of intrapulmonary regulatory peptides on the expressions of HLA-DR,CD80 and CD86 in human bronchial epithelial cells;
肺内调节肽对人支气管上皮细胞HLA-DR、CD80、CD86表达的影响
4) Human bronchial epithelial cell
人支气管上皮细胞
1.
Malignant transformation of human bronchial epithelial cells induced by benzo(a)pyrene metabolite dihydroxyepoxy benzo pyrene;
苯并(a)芘代谢物反式二羟环氧苯并芘诱发人支气管上皮细胞恶性转化
2.
Objective To explore the mechanisms of chemical carcinogenesis and the relationship between malignant transformation and genomic/chromosomal instability of human bronchial epithelial cell line by formaldehyde.
目的为探讨化学致癌的机制,揭示恶性转化与染色体不稳定性之间的关系,本研究考察甲醛对人支气管上皮细胞系转化和染色体稳定性的影响。
3.
Purpose: To subclone the malignant transformants of human bronchial epithelial cell line (BEP2D) induced by α-particles and to analyse their karyotypes and capacity of rejoining DNA double-strand breaks(DSB).
目的 :建立α 粒子诱发人支气管上皮细胞 (BEP2D)恶性转化细胞的克隆细胞系 ,研究其核型和DNA链断裂修复能力。
6) Human bronchial epithelial cell line
人支气管上皮细胞
1.
Purpose: To study the malignant transforming activity of crystalline nickel sulfide (NiS) in human bronchial epithelial cell line (16HBE) immortalized by SV-{40} Large T antigen.
目的 :研究结晶型硫化镍 (NiS)诱发SV4 0 LargeT抗原永生化的人支气管上皮细胞系 (16HBE)恶性转化作用。
2.
[Objective] To investigate the influence on apoptosis of human bronchial epithelial cell line(BEAS-2B)induced by chrysotile fibers and to compare the methods of single cell gel electrophoresis assay(SCGE)with flow cytometry analysis(FCM)for detecting apoptosis.
[目的]研究温石棉对永生化人支气管上皮细胞(BEAS-2B)凋亡的影响,比较单细胞凝胶电泳试验(SCGE)与流式细胞术(FCM)检测细胞凋亡结果的异同。
补充资料：急性喉气管支气管炎
急性喉气管支气管炎
〖HT5”SS〗acute laryngotracheobronchitis
上、下呼吸道的急性弥漫性炎症，以喉部及声门下的浮肿、气管与支气管内渗出物稠厚成痂，以及中毒现象为特征。可为流行性或散发性，往往继发于麻疹或流行性感冒之后。多见于3～5岁的幼儿，因此时对传染病抵抗力低，咳嗽功能差，加之分泌物黏稠不易咳出，更助长感染的蔓延。冬季与早春气候干燥时发病较多。患处黏膜充血肿胀，可见糜烂或溃疡，甚至侵入肌层，以致管腔狭窄。
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
参考词条