1) Systemic inflammatory response syndrome Multiple organ dysfunction syndrome Perinatal factors Neon(
全身炎症反应综合征多器官功能不全围产因素新生儿
3) MODS
多器官功能不全综合征
1.
The insulin resistance and the changes of density and affinity of insulin receptor on erythrocytes surface in MODS patients;
多器官功能不全综合征患者胰岛素抵抗及胰岛素受体密度和亲和力变化的研究
2.
Objective To observe the changes of intestinal microflora,proliferation of GNB,intestinal free endotoxin,bacterial translocation to the MLN and their relationships in the MODS rats.
目的探讨多器官功能不全综合征(MODS)大鼠肠道细菌微生态的变化及其与肠源性内毒素血症和细菌易位的关系,并观察大承气汤的影响。
3.
Objective To establish the improved diagnostic standards of the trauma MODS involved Islet β-cell dysfunction and to diagnosis the trauma MODS early.
目的制订涉及胰岛β细胞功能不全的创伤性多器官功能不全综合征(MODS)诊断方案,早期诊断和发现创伤MODS。
5) Multiple organ dysfunction syndrome
多器官功能不全综合征
1.
Establishment of Rat Model of Multiple Organ Dysfunction Syndrome Caused by Trauma;
严重创伤多器官功能不全综合征大鼠模型建立
2.
The concept of"bacterial translocation" once explained the potential role of gut in the development of multiple organ dysfunction syndrome(MODS)and elicited the term of "gut barrier".
细菌移位的提出揭示了肠道在多器官功能不全综合征(MODS)中的重要作用,并引出肠屏障的概念。
3.
Objective To investigate effect of Dachengqi(大承气)Granula treatment to morphology of interstitial cells of Cajal(ICC)in rat gastrointestine in multiple organ dysfunction syndrome(MODS)and to find out the mechanism of the drug Methods Twenty Wistar rats were randomly divided into two groups,the MODS group and the group which were treated with Dachengqi granula.
目的:研究大承气冲剂在治疗多器官功能不全综合征(MODS)过程中对Cajal间质细胞(ICC)形态学变化的影响,探讨药物作用机制。
6) systemic inflammatory response syndrome (SIRS)
全身炎症反应综合征
1.
Correlation of Systemic Inflammatory Response Syndrome (SIRS) with Death of Cholangitis-originated Shock in the Aged;
全身炎症反应综合征与老年胆源性休克病人死亡的相关性调查
2.
Objective To observe the intervention of systemic inflammatory response syndrome (SIRS) at early stage in newborn infants with low-dose hepsrin.
目的观察小剂量肝素对危重新生儿全身炎症反应综合征(SIRS)的治疗效果及预后的影响。
补充资料：全身性炎症反应综合征
全身性炎症反应综合征
机体对各种严重损伤，包括感染、创伤、烧伤、缺氧和再灌注等引起的全身反应。它不是一种新发现的疾病，而是基于对感染、炎症和严重损伤发生、发展机制深入认识，原于20世纪90年代初由美国胸科医学会和危害急救医学会提出的新概念。这种反应有下列四种表现：体温升高或降低，心率和呼吸增快，白细胞增高或减少、分类左移。炎症介质失控性释放是其主要发生机制。其特征是全身的高代谢反应，表现为高通气量、高血糖、蛋白分解增加和高乳酸血症和体内炎症介质浓度增高。早期预防、诊断和治疗有利于降低小儿SIRS发病率和病死率。
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
参考词条