1) left-side-portal hypertension
左侧门脉高压症
1.
Objective To analyze 12 left-side-portal hypertensions(LSPH) misdiaghosed as cirrhosis and point out the essentials of diagnosis and differential diagnosis between LSPH and cirrhosis.
目的：通过对左侧门脉高压症误诊肝硬化１２例临床分析，指出其诊断要点及与肝硬化的鉴别诊断。
2) Portal hypertension
门脉高压症
1.
Experimental study of subtotal splenectomy for treating portal hypertension;
脾次全切除术治疗门脉高压症的实验研究
2.
The therapeutic effects of portal aterialization and complete shunt in portal hypertension dogs;
脾动脉门静脉吻合联合完全门体分流对犬门脉高压症的治疗观察
3.
Surgical treatment for upper gastrointestinal rebleeding after pericardial devascularization in portal hypertension patients;
门脉高压症断流术后上消化道再出血的外科治疗探讨
3) portal hypertension
门静脉高压症
1.
Tunnel pericardial devascularization on upper gastrointestinal bleeding for portal hypertension patients:an analysis of 307 cases;
隧道式贲门周围血管离断术治疗门静脉高压症307例疗效分析
2.
Akt gene therapy for cirrhotic rats with portal hypertension;
Akt基因治疗大鼠肝硬化门静脉高压症
3.
Treatment of patients with portal hypertension and calculus of bile duct;
门静脉高压症与胆道结石关系的处理
4) protal hypertension
门静脉高压症
1.
Objective: To investigate the expression and clinical significance of CD40 and Caspase-9 in the spleen of cirrhotic protal hypertension.
方法：采用免疫组织化学S-P法测定50例肝硬化门静脉高压症患者脾脏和15例正常脾脏中CD40及Caspase-9的表达情况。
2.
Objective: By analyzing the expression of HLA-ABC and HLA-DR in the spleen of patients with cirrhosis and protal hypertension, and the relationship between the expression and some correlated clinical parameters, studied the possible mechanism of the splenic immune function change in those patients.
目的:通过分析HLA-ABC、HLA-DR在肝硬化门静脉高压症脾脏中的表达情况及其与相关临床参数的关系,研究肝硬化门静脉高压症时脾脏免疫功能改变的可能机制。
5) Pancreatogenous portal hypertension
胰源性门脉高压症
1.
Pancreatogenous portal hypertension in 19 cases;
胰源性门脉高压症19例
6) Idiopathic portal hypertension
特发性门脉高压症
1.
Coagulation indexes in patients with idiopathic portal hypertension(IPH) or hepatic cirrhosis(HC) were studied.
研究特发性门脉高压症(IPH)和肝硬化患者的凝血酶原时间(PT)和凝血酶原标准化比率(INR)、活化部分凝血活酶时间(APTT)和纤维蛋白原(Fib)。
补充资料：门脉高压症
门脉高压症
系指肝脏门静脉系统压力升高，主要由于肝内正常结构被改建，血流阻力增高引起，常见于肝硬化。门脉压升高以后，胃、肠和脾等器官的静脉血回流受阻，早期因代偿可无明显症状，晚期常表现为脾肿大和脾功能亢进，胃肠道瘀血，腹水，侧支循环的建立与开放。如果食管静脉丛发生破裂，可引起呕血、黑便。
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
参考词条