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1)  extracorperal artificial liver
体外人工肝
2)  Extracorporeal bioartificial liver support system (EBLSS)
体外生物人工肝支持系统
3)  artificial liver
人工肝
1.
Application of comfortable nursing in the treatment of serious hepatitis with artificial liver;
舒适护理在人工肝治疗重型肝炎中的应用
2.
Clinical application of non-bioartificial liver to patients with chronic severe hepatitis;
非生物人工肝治疗慢性重型肝炎的临床应用
3.
Clinical study on novel artificial liver(albumin dialysis) in severe hepatitis;
新型人工肝治疗重型肝炎的临床研究
4)  Artificial liver support system
人工肝
1.
Analysis of Prognosis Factors of Artificial Liver Support System on Patients with Severe Hepatitis;
人工肝支持系统对重型肝炎预后的影响因素分析
2.
This article introduces the appropriated indications and rationalized selection of artificial liver support system, the present situation on study of bioartificial liver, and the possible combination of artificial liver and liver transplantation according to the clinical experience of the Drum Tower Hospital.
 结合临床经验介绍人工肝应用中适应症选择、具体技术合理化应用、生物人工肝研究现状和人工肝联合肝移植治疗肝衰竭。
3.
Objective Explore heavy hepatitis patients who accepted artificial liver support system treatment in order to enhance patients who accepted artificial liver support system treatment salvage success rate and provide credible cure foundation to clinic.
目的探讨人工肝血浆置换术患者医院感染因素及预防方法,提高重症肝炎患者抢救成功率,为临床提供可靠治疗依据。
5)  Molecular Adsorbents Recirculating System
MARS人工肝
1.
Objective:To summarize and evaluate the treatment effective and mechanism of Molecular Adsorbents Recirculating System in patients with severe hepatitis.
目的:探讨和评价MARS人工肝在重症肝炎治疗中的疗效及其作用机理。
2.
Objective To evaluate the chang es of hemodynamics and the relationship with the outcome in patients with severe h epatitis treated with molecular adsorbents recirculating system (MARS).
方法 应用MARS人工肝支持治疗重症肝炎 16例共 36次作为治疗组 ,对照组为 2 0例重症肝炎 ,采用综合治疗 ,两组年龄、性别及肝功能相匹配 ;观察单次治疗前后平均动脉压、心率的变化及与主要生化指征和预后的关系。
6)  Artificial liver
人工肝脏
补充资料:肝外阻塞性胆汁性肝硬化


肝外阻塞性胆汁性肝硬化


又称"继发性胆汁性肝硬化。*胆汁性肝硬化的一个类型,继发于肝外胆管阻塞而引起的肝硬化。主要由总胆管结石、手术后胆管狭管、胰头癌、先天性胆管闭锁或缺如等原因所造成。在梗阻上端的胆道由下而上地逐渐扩大迂曲,胆汁淤积,肝内小胆管扩大而破裂,使肝血管受压,胆汁外渗,肝细胞发生缺血、坏死,纤维组织增生,最后形成肝硬化。临床表现为黄疸、肝脾肿大,皮肤粘膜出血倾向,血胆固醇下降等,晚期表现同*肝硬化。解除肝外阻塞原因,本病有治愈的可能性。
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