1) posthepatitic B cirrhosis
乙型肝炎性肝硬化
1.
To observe the changes of plasma peptide hormone levels in patients with posthepatitic B cirrhosis, and discuss their influence on hemodynamic as well as retention of sodium and water, RIA method was used to determine the plasma hormone levels in the patients with cirrhosis and in healthy control group.
采用放射免疫分析法测定空腹血浆的激素含量 ,观察乙型肝炎性肝硬化时血浆肽类激素变化 ,探讨其对血液动力学和钠水潴留的影响。
2) Hepatitis B Cirrhosis
乙型肝炎肝硬化
1.
Effects of rocuronium on the onset time of muscular relaxation in patients with hepatitis B cirrhosis;
罗库溴铵对乙型肝炎肝硬化患者肌松时效的影响
3) Liver cirrhosis
乙型肝炎肝硬化
1.
[Objective] To investigate whether HLA class II DQA1 and DQB1 gene polymorphisms are associated with development of chronic hepatitis B virus (HBV) infection in Shandong province and provide clues to seeking for the susceptible and protective genes for chronic HBV infection, HBV-related liver cirrhosis (LC), and hepatocellular carcinoma (HCC).
目的探讨山东地区人类白细胞抗原(human leucocyte antigens,HLA)DQA1、DQB1基因的等位基因多态性与慢性乙型肝炎病毒(hepatitis B virus,HBV)感染及其临床转归的相关性,为寻找慢性乙型肝炎病毒感染、乙型肝炎肝硬化及乙肝后肝细胞癌的易感基因和抗病基因提供线索。
4) HBV-hepatic cirrhosis
乙型肝炎肝硬化
1.
Objective: To investigate the relationship between Genetic polymophisms ofConnective tissue growth factor promoter and HBV-hepatic cirrhosis Methods: Genetic polymorphisms of connective tissue growth factorpromoter (locus-484G/C and-650G/C) were determined in 121 patients withHBV-hepatic cirrhosis and 138 healthy blood donors by polymerase chainreaction (PCR) and direct capillary sequencing.
目的:研究结缔组织生长因子(connective tissue growth factor,CTGF)基因启动子多态性及其与四川地区中国人乙型肝炎肝硬化发生、发展的相关性。
2.
Objective: To investigate the relationship between genetic polymorphism in angiotensin-converting enzyme (ACE), angiotensin II type 1 receptor (AT1R) and HBV-hepatic cirrhosis.
目的:研究血管紧张素转换酶(angiotensin-converting enzyme,ACE)及血管紧张素Ⅱ1型受体(angiotensinⅡtype 1 receptor,AT1R)基因多态性与乙型肝炎肝硬化之间的关联,旨在初步探讨其在乙型肝炎肝硬化的发生、发展中的作用。
5) Decompensated hepatitis B cirrhosis
乙型肝炎肝硬化代偿期
补充资料:肝外阻塞性胆汁性肝硬化
肝外阻塞性胆汁性肝硬化
又称"继发性胆汁性肝硬化。*胆汁性肝硬化的一个类型,继发于肝外胆管阻塞而引起的肝硬化。主要由总胆管结石、手术后胆管狭管、胰头癌、先天性胆管闭锁或缺如等原因所造成。在梗阻上端的胆道由下而上地逐渐扩大迂曲,胆汁淤积,肝内小胆管扩大而破裂,使肝血管受压,胆汁外渗,肝细胞发生缺血、坏死,纤维组织增生,最后形成肝硬化。临床表现为黄疸、肝脾肿大,皮肤粘膜出血倾向,血胆固醇下降等,晚期表现同*肝硬化。解除肝外阻塞原因,本病有治愈的可能性。
说明:补充资料仅用于学习参考,请勿用于其它任何用途。
参考词条