1)  multidrug resistance
多药耐抗药性
2)  multiple
多药
1.
Chemoprotective role of multiple drug resistance gene over expression in human bone marrow CD34~+ cells;
多药耐药基因在人骨髓CD34~+细胞中的表达及对化疗药物的耐受性
2.
Relationship between expression of pS2, ER, PR, c-erbB-2 and multiple drug resistance in breast cancer;
乳腺浸润性导管癌pS2基因、ER、PR、c-erbB-2与多药耐药基因表达及临床指标的相关性
3.
Objective: To evaluate the feasibility of gene therapy using Zinc ribbon domaincontaining 1 (ZNRD1) as target for multiple drug resistance of leukemia.
目的:探讨转录相关锌带蛋白基因(zincribbondomaincontaining1protein,ZNRD1)作为靶分子进行白血病多药耐药基因治疗的可行性。
3)  Multidrug
多药
1.
Reversal of multidrug resistance of K562/A02 cell line by mdr1 and GSTπ gene silence;
mdr1和GSTπ基因缄默逆转K562/A02细胞多药耐药性的研究
2.
Progress in the study for mechanisms of multidrug resistance in breast cancer;
乳腺癌多药耐药机制研究进展
3.
OBJECTIVE: To evaluate the relationship between the clinical stages of nasopharyngeal carcinoma (NPC) and the expression of multidrug resistance-associated protein (MRP).
目的:探讨多药耐药相关蛋白(mul-tidrugresistance-associatedprotein,MRP)在鼻咽癌中的表达及与临床分期的关系。
4)  multidrug resistance
多药耐药
1.
Function and mechanism of Baihua Sheshecao Injection reversing multidrug resistance of K562/ADM cells;
白花蛇舌草注射液逆转K562/ADM细胞多药耐药的作用和机制
2.
In Vitro study of the effect of cyclosporine A,tetrandrine and their combination on the reversion of multidrug resistance in leukemia cell line;
环孢菌素A、汉防己甲素及两者联合逆转K562/A02细胞的多药耐药
3.
Reversal of multidrug resistance of hepatocellular carcinoma by siRNA/mdr1;
RNA干扰逆转肝细胞癌多药耐药
5)  multi-drug resistant
耐多药
1.
Fiberoptic bronchoscopic interventional therapy of multi-drug resistant caseous tuberculous pneumonia with gel containing drugs;
含药凝胶经纤维支气管镜介入治疗耐多药结核性干酪性肺炎
2.
Efficacy of freeze-dried M. vaccae vaccine in treatment of patients with multi-drug resistant pulmonary tuberculosis;
冻干治疗用母牛分支杆菌菌苗对耐多药肺结核患者临床疗效及细胞免疫功能的影响
3.
Experience in treating 86 patients with multi-drug resistant pulmonary tuberculosis by used ALP ( amikacin, levofloxacin, sodium, aminosalicylate ) regimen;
ALP方案治疗86例耐多药结核病的疗效分析
6)  Multi-drug resistance
多药耐药
1.
The research of CIK cells reverse multi-drug resistance in vitro;
CIK细胞体外逆转多药耐药性的研究
2.
Relation between multi-drug resistance and apoptosis of HepG2/5-Fu;
人肝癌HepG2/5-Fu耐药细胞株多药耐药与凋亡关系的研究
3.
Expression of glutathione S-transferase π and multi-drug resistance associated protein in squamous cell carcinoma tissue;
皮肤鳞状细胞癌组织中谷胱甘肽-S-转移酶和多药耐药相关蛋白的表达
参考词条
补充资料:抗真菌药和抗立克次氏体药


抗真菌药和抗立克次氏体药
Chemotherapeutics, Antimycotic and Antirickettsial

kans抗真菌药和抗立克次氏体药第9卷 O }}(1)CICH之一C一CHZCI一-~甲~~~~~叫~~一~~~(2)CHaC00H FB了IJ ︸胜、!尸以.纵扮!F(48) OH lCICH:一C一CHZCIN=,、 1,1 NH尸丫r,一一三之~、汗尸Na月,DMF F(49)(11) :HzN~3。:。您丫督自气丛立竺竺自护、-跳沪/、‘犷HCOH、屯/卜‘2 (51)(52) 。之丫⑧ 坐助OH(‘2,(53) HCI-~~,~~~~~~,..~ 一H20‘HCI(90)侧 月F C.\H一\l尸n下.C上z、丫F H C 一 N =‘ N 1.1七(47) 氟康哇口服吸收快而完全,半衰期长(22~32h)体内分布均匀,能很好的透过血脑屏障,并渗透到脑脊液内阳〕。口服100mg后,脑脊液平均浓度为血浆浓度的0.58一0.89倍。70%原药由肾脏排除。 大多数病人对氟康哇耐受良好哪〕。常见的副作用为头痛、恶心、疲劳、发热、浮肿、出疹、腹部不适及转氨酶升高。但发生率小于5环,极少数病人出现血小板减少,停药后即恢复。·2.8.烯丙胺类 1981年发现蔡替芬(haftifine)(s0)具有较高的广谱抗真菌活性。由于其优良的抗真菌活性,新颖的结构特点,很快引起了人们的重视,通过对其结构改造和抗菌活性的研究,发现了活性高、毒性低的衍生物特比禁芬(terbinafine)(‘oa)和丁禁芬(botenafine)(乐ob)。活性最大,最低抑菌浓度(Mlc)为。.01一0.2哆/ml,对申克氏抱子丝菌和曲霉属真菌次之,Mlc分别为0.8一1.5限/耐和0.8~12.5阳/而。禁替芬的体外抗皮肤真菌活性明显优于咪哇类抗真菌药,而与托蔡醋(发癣退)和灰黄霉素相似。 蔡替芬对真菌细胞超微结构有影响,还干扰真菌细胞的脂质代谢,故有杀菌作用。在作用机理上蔡替芬与氮哇类药物一样,都是抑制麦角幽醇的生物合成,但作用部位二者不同,禁替芬是角拨烯的环氧化酶的特异性抑制剂。通过抑制角鳖烯的环氧化,使角盆烯蓄积和麦角幽醉缺乏,使真菌细胞膜的组成和通透性发生改变,导致杀菌作用[5,〕。 蔡替芬是一个高效低毒的外用抗真菌药,其疗效与克霉哇、美康哇、益康哇等外用抗真菌药相似。
说明:补充资料仅用于学习参考,请勿用于其它任何用途。