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1)  model of renal hypertention with complication of hyperlipemia
肾性高血压并高血脂模型
2)  renal hypertension rats
肾性高血压大鼠模型
1.
Objective To observe the effect of Shouwujiangya (SWJY) capsule on blood pressure, urinary content of immune globulin G (IgG), α1-microglobulin (α1-MG) and β2-microglobulin (β2-MG) of the renal hypertension rats (RHR).
方法采用二肾一夹(2K1C)法复制肾性高血压大鼠模型,观察尿中IgG、α1-MG、β2-MG的含量。
3)  2K1C renal hypertension rats
2K1C肾性高血压大鼠模型
4)  Renal hypertension
肾性高血压
1.
Influence of Shouwujiangya Capsules on plasma 6-Keto-PGF_(1α) and TXA_2 in rats with renal hypertension;
首乌降压胶囊对肾性高血压大鼠血浆前列腺素、血栓素水平的影响
2.
Effects of Sesamin on Contraction of Aortic Ring,NO and ET in Renal Hypertension Rats;
芝麻素对肾性高血压大鼠主动脉收缩功能、NO和ET含量的影响
3.
Effects of Fufang Jiangya Pian (FFJYP,复方降压片) on renal hypertension rat;
复方降压片对肾性高血压大鼠模型的影响
5)  renovascular hypertension
肾性高血压
1.
Methods A total of 24 healthy female Wistar rats were randomly divided into sham operation(SO)group,renovascular hypertension(RH)group and SND treatment(ST)group consisting of 8 cases in each group.
目的探讨四逆汤对肾性高血压大鼠血液内血管活性物质含量和主要靶器官血管活性物质表达的调节作用。
2.
METHODS 2 kidney-1 clip (2K1C) renovascular hypertension was induced by subjecting the SD rats to left renal artery clamping.
方法 通过缩窄左肾动脉而制备2K1C夹的肾性高血压模型,同时选用16周龄的SHR ,2种实验鼠均连续给予Qi盐0 。
6)  renovascular hypertensive
肾性高血压
1.
Influences of ZiShuJiangHuoYing(ZSJHY) on the Levels of Serum Cytokines in Renovascular Hypertensive Rates(RHR);
滋水降火饮对肾性高血压大鼠血清细胞因子水平的影响
2.
The renovascular hypertensive rats(RHR) and spontaneously hyper tensive rat (SHR) were given po the decotion of total alkaloids of Uncar ia rhychophylla (Tau mw) 30~7.
5 mg/kg给肾性高血压大鼠(RHR)和自发性高血压大鼠(SHR)灌胃后,通过清醒大鼠尾动脉间接测血压法观察血压(BP)变化。
3.
Objective: To investigate the effects of perindopril with spironolactone on myocardial fibrosis and expression of transforming growth factor-β_1(TGF-β_1) and matrix metalloproteinase 2(MMP-2) in renovascular hypertensive(RH)rats.
目的:探讨转化生长因子-β_1和基质金属蛋白酶-2在肾性高血压大鼠心肌纤维化中的作用以及培哚普利联合螺内酯的干预效果。
补充资料:理糖激素反应性高血压


理糖激素反应性高血压


  病名。即17-α羟化酶缺乏综合征。详见该条。
  
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