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1)  Mismatch repair enzyme
错配修复酶
2)  DNA mismatch repair enzyme
DNA错配修复酶
1.
Construction of hMSH2-deficient cell strain of DNA mismatch repair enzyme;
DNA错配修复酶hMSH2缺陷细胞株的建立
3)  Mismatch repair
错配修复
1.
Study on mismatch repair genes of chronic myeloid leukemia;
慢性粒细胞白血病错配修复基因的研究
2.
Genomic instability and mismatch repair mechanism;
基因组不稳定性与错配修复机制
3.
coli helicaseⅡ(UvrD),which is classified as a 3 to 5 helicase,plays essential roles in methyl-directed mismatch repair(MMR)and nucleotide excision repair(NER).
coli解旋酶Ⅱ(UvrD)是一种在甲基定向错配修复(methyl-directed mismatchrepair,MMR)和核苷酸切除修复(nucleotide excisionrepair,NER)中起重要作用的3′→5′解旋酶。
4)  DNA mismatch repair
DNA错配修复
1.
Methylation of DNA mismatch repair gene MLH1 and MSH2 in acquired multidrug-resistance of human small cell lung cells H446;
DNA错配修复基因甲基化在H446细胞获得性耐药中的作用
2.
Advances of researches on the composition and function of DNA mismatch repair system;
DNA错配修复系统组成和功能的研究进展
3.
Frequent Ki-ras Mutation in MSI-H Colorectal Cancer Deficient in DNA Mismatch Repair Genes;
DNA错配修复基因缺陷的MSI-H大肠癌频发Ki-ras基因点突变
5)  Mismatch repair genes
错配修复基因
1.
Effects of 5-aza-2 -deoxycytidine on apoptosis of ovarian cancer cells and on expression of mismatch repair genes;
5-氮-2′-脱氧胞苷对人卵巢癌细胞凋亡及DNA错配修复基因表达的影响
2.
Large genomic deletions of mismatch repair genes in Chinese patients with hereditary nonpolyposis colorectal cancer;
中国人遗传性非息肉病性结直肠癌错配修复基因大片段缺失研究
3.
Large deletion in mismatch repair genes uncovered by quantitative multiplex PCR-high performance liquid chromatography system;
定量多重PCR-高效液相色谱分析错配修复基因大片段缺失
6)  Mismatch repair gene
错配修复基因
1.
Relationship of mismatch repair genes hMLH1 and hMSH2 expression with cell proliferation in colorectal adenomas;
大肠腺瘤及其癌变错配修复基因hMLH1和hMSH2表达与细胞增殖的关系
2.
Study of relationship between expression of DNA mismatch repair gene hMSH_2 and generation of gastric cancer;
DNA错配修复基因hMSH_2蛋白表达与胃癌发生关系的探讨
3.
Effects of cadmium stress on expression of mismatch repair gene in Arabidopsis thaliana seedlings;
镉胁迫对拟南芥幼苗错配修复基因表达的影响
补充资料:光致酶修复
分子式:
CAS号:

性质:紫外线照射可以使DNA分子中同一条链的两个相邻嘧啶碱基之间形成二聚体,特别在胸腺嘧啶之间。嘧啶二聚体的形成,影响了DNA双螺旋结构,使其复制和转录功能受到阻碍。DNA受损伤部分,可通过光复活作用而修复:而光复活酶结合于损伤部位,酶被可见光激活,修复损伤部位,酶脱离修复损伤部位,此为修复全过程。

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